Medical Stupidity

Epiphenom: The doctors who hasten death

2010-03-08T13:25:00.000-08:00

Epiphenom: The doctors who hasten death

Articlesbase: Code Zero

The Arlene Berry Story

2009-05-18T14:38:00.000-07:00

In
Memory Of
Arlene
H. Berry
1958-2000

The implications of the findings herein are so
profound that, according to a hospital insider, Health Canada has
issued a "gag-order" in an effort to save face (with
so many doctors and nurses involved in this unnecessary death), a
blatant attempt not only to prevent the media from becoming involved,
but also as a means to deny justice. In my opinion the health
authorities have also insinuated themselves into a conspiracy, or
party accessory (after the fact) to a medical homicide. These
political and ministerial thugs, attached to the public purse,
including the sinister College of Physicians and Surgeons of Ontario
and their corporate counterparts seem to think that they and the
physicians they seek to shield are all above the law.

Published under a wide variety of titles
internationally, the Arlene Berry case stands as a testament to what
can only be construed as widespread corruption in Ontario's
health-care system.

In Memory of Arlene Berry

The FACTS: Update 2009

In December of 1999, Arlene Berry was sent to Timmins
& District Hospital in Timmins, Ontario, where she was diagnosed,
according to her physician, with "carcinoma of the left main
bronchus with residual cancer of the aorta due to a complete collapse
of the left lung".

Her family MD, Dr. Edward. H Jordan had been treating
her assumptively for what he termed a "suspected bronchitis".
It took another doctor to read her X-ray chart, and to order more
appropriate testing before anything was done.

On or about January 12th of 2000, Arlene Berry was
admitted to the Timmins & District Hospital in Timmins, Ontario,
where she had a left lung pneumonectomy on January 13th of
2000, under the care of Dr. Claudio de la Rocha, a Cardiovascular and
Thoracic Surgeon who immigrated to Canada from Mexico.

Following surgery, Arlene Berry was discharged home 5
days later. On or about March 16thof 2000, Arlene Berry returned to
Timmins where she underwent follow-up study and testing at the same
hospital, consisting of a CT scan, and a mediastinoscopy with
mediastinotomy, as part of her post-operative evaluation.

What the family had found to be peculiar however, was
a dramatic voice change at some point following the mediastinal
procedures, suggesting a "partial vocal fold paralysis"
thought to have been procedure related. Although she began to regain
her voice in the weeks that followed, her voice remained somewhat
"whispery" for the remainder of her days.

Following that testing, Arlene confided "I don't
have AIDS, or brain tumors, or anything like that, but I might have a
"cyst", or "infection".

A cystis a suitcase for the infectious material
inside. Some of them parasites with simulation of stroke related
damage in cultured human nerve cells have been reported. Trapped
blood (ie. from an old hemorrhage or occipital bleed) can also lead
to the growth of cysts. Patients can be asymptomatic even if the cyst
is quite large.

Arlene Berry was then referred to
the Northeastern Ontario Regional Cancer Centre situated at the
Laurentian Site, Sudbury, Ontario for consideration of radiation
therapy, under the care of Dr. Hugh Prichard, a radiation oncologist.
By the end of April 2000, Arlene Berry had completed her
post-operative course of radiation therapy. In light of this
treatment, her condition was seen to be stable. She had been
scheduled for X-ray follow-up in Sudbury on Tuesday May 30th at 2:30
PM.

Following radiation, Arlene Berry developed
"flu-like" symptoms suggestive of gastrointestinal
illness within two weeks following radiation therapy, at the
end of April of 2000. She died on the 24th of May of 2000.
Total time lapse 24 days; mean 3 weeks plus, or just under 4 weeks.

I have given
careful consideration to Guillain Barre syndrome. This disorder can
develop over the course of hours or days, or it may take up to 3 to 4
weeks. Most people reach the stage of greatest weakness within the
first 2 weeks after symptoms appear. 66% reach nadir in 2 weeks, 92%
in 3 weeks; by definition MUST peak at 4 weeks

According to her Rx scripts, Arlene Berry had
been given Amoxicillin for infection. Amoxicillan belongs to a class
of penicillin-like drugs, side effects of which include severe nausea
and vomiting, including abdominal pain.

According to the
hospital record Arlene Berry was admitted to the Kirkland and
District Hospital on May 23rd of 2000 by Dr. Spiller for "IV
fluid and Gravol", evidenced at A-6. According to record, she
was given more than IV and Gravol. If not Dr. Spiller, who ordered
the 30 mg MS Contin on his watch? From that record it seems clear
that either Dr. Spiller lied, or that he was totally oblivious to the
administration of Morphine Sulfate evidenced at A-12. According to
the same record, she was admitted for "vomiting".

Vomiting is not a diagnosis, but
rather a symptom of many causes.

A-5 of the record documents the presenting complaint
as "headaches, accompanied by severe stomach pain",
and "abdominal pain ongoing for 2 weeks", for which
she had been prescribed "antibiotics".

Abdominal or stomach pain
concurrent with nausea and vomiting points to the abdomen as
the source of the problem, which should have been a 'red flag'
suggesting the possibility of intestinal
obstruction. Abdominal pain can also be the
result of "intestinal ischemia". The hallmark of intestinal
ischemia is "abdominal pain".

OP-53
documents a history of bloody bowel movements
when voiding evidenced by "bloody BM's x
4 days" (bloody, black, or tarry
stools). Bloody stools may signify bleeding stomach, diverticular
bleeding, or intestinal infection. The same record documents
"pale-looking and lethargic".
Pale skin suggests decreased blood supply
to the skin. Blood vessels in the body constrict to conserve blood in
the body's core, making you feel cold and your skin go pale. Lethargy
and drowsiness are often associated with
moderate to severe dehydration,
including congestive heart failure. Lethargy may also be caused by
the toxic effects of
waste products on brain function.

According to the record at
OP-53 she was "Here 1 week ago for UTI.
Last period on 6th of May". Onset of
menstrual periodis closely related to onset of migraine headaches,
including illness. Case reports cited primarily in women having
period in which a blood-soaked tampon may provide an excellent
breeding ground for the bacteria and is a significant cause of
urinary tract infections. The same record dated May 22nd documents
"For 2 weeks had flu, migraines".
Flu symptoms — like headache, fever, chills, stomach pain, usually
appear anywhere from 1 to 4 days after a person has been exposed to
the virus. Flu headache may be caused by swelling of blood vessels in
the brain, due to increasing pressure. Headaches from the flu can
feel a lot like migraines. Influenza is not the same as a “stomach
flu,” This is most likely due to a GI (gastrointestinal)
infection—not the flu. Headaches are also common in people with GI
trouble. Many different types of bacteria and parasites can also
cause GI infections, leading to CNS infection.

According to the record at A-6, she returned
to the ED (emergency department) the following day, on May 23rd of
2000 "with the very same
complaints".

Rapid evolution of illness and
patient return within 24-48 hours suggests a severe illness.

The RN who saw her noted that she had been "taking
morphine" for pain management, and also that she had
recently "stopped" taking the morphine, noting her
recent medical history that for "2 weeks" she had
the "flu". The same record also documents a question
mark (?) with respect to possible morphine allergies, seen at
A-5.

OP-53 documents a history of Tylenol and Aspirin,
including a documented "daughter states takes a lot",
suggests a history of drugs that can break the gastric barrier, and
damage the gastric mucosa, ie., NSAID's (non-steroidal
anti-inflammatory drugs). Aspirin is the drug classically associated
with Reye syndrome.

According one of her Rx scripts, Arlene Berry had
been prescribed sodium phosphate
(usually used to treat constipation) while under the care of her
oncologist, and sodium dosucate
prescribed by her family MD, and to the best of my knowledge kept
taking them following her discharge home, until her prescription ran
out. She had found the prescribed laxatives to be ineffective and so
turned to over the counter laxative and tap water enemas for what
appeared be drug opiate induced dysmotility.

Constipation,
fecal impaction and bowel obstruction are also common problems for
oncology patients.

Enema would have been contraindicated, especially in
patients with fulminant disease, because of the danger of
precipitating toxic megacolon or perforation of the colon.
Unfortunately, the health care providers who attended to this patient
failed miserably to educate or instruct her in helpful ways.

What appears to be a referral at A-6 of the medical
record, a chart-copy from the admitting physician (Dr. Spiller),
directed to the attention of the family physician, Dr. Jordan,
documents what I take to be a provisional diagnosis of "vomiting",
while the record at N-11 documents "vomiting, lung CA".

According to Dr. Jordan, "she had presented to
the ED several days before with vomiting and it was thought that she
had a UTI", to rule out delay in seeking treatment. According to
the hospital record at A-8 "she was given antibiotics and
sent home".

According to the outpatient record at OP-54, the
patient's recent head CT scan showed "NO METASTASIS",
and her mediastinoscopy, a surgical procedure to examine the
mediastinum inside of the upper chest between and in front of
the lungs, were found to be "NEGATIVE". From that
record it seems clear that NO clinically detectable metastasis
were found.

The Health Management Record at A-21 of the
record documents the patient's sensory cognitive perceptual pattern
as "sedated". Increased sedation is also a serious
side effect of many pharma agents, including electrolyte derangements
which can mimic sedative intoxication.

The record at A-23 documents a "slurred
speech" as evidenced by a checkmark in the upper left
corner. Speech that is unintelligible, slurred, or whispery,
suggests that the various muscles required to form speech are
weakened.

The record at OP-54 dated May 22nd of 2000
documents a "haggard appearance", including "large
blood trace leukocytes", what I take to mean leukocyte
estrace, marked by an unusually high number of white blood cells
(WBC's).

Weakness of facial muscles
produces a characteristic haggard appearance,
or a deceptively disinterested facial expression. The characteristic
appearance of a "haggard" or "mournful" face and
drooping eyelids is caused by facial muscle
paralysis. A sagging mouth or a crooked smile
is a part of the same problem.

The Outpatient Record from the
hospital dated May 22nd of 2000, seen at OP-54 documents a recent
history of "hematuria" (blood
in urine) for "three days".
The healthcare provider who saw her made a diagnosis of UTI.
The same record documents a prescription for Cipro,
for treatment of "urinary-tract
infection". The belated test result
however, what I assume to have been a urology test, or a bacterial
culture test, evidenced at OP-55 of the record, later returned a
finding of "NO Growth".
A negative urine test can also suggest the presence of unusual
bacteria or viruses causing symptoms of UTI.

The record at OP-54 documents "SEPTRA DS
GIVEN BEFORE & CIPRO GIVEN AFTER". The same healthcare
provider (whose signature is illegible) also made a notation with
respect to the "flu", which was then directed to the
attention of the patient's "family MD", namely, Dr. Jordan.

Cipro is a broad-spectrum antibiotic indicated
in the treatment of a variety of infections, including the flu.
Bactrim/Septra is also the antibiotic most frequently
associated with drug-induced aseptic meningitis. Certain combinations
of medications, such as penicillin and sulfa-based antibiotics
can cause the body's immune system to react by over-stimulation,
sending white blood cells (called T cells) rampaging through the body
destroying its own tissues. This is known as an autoimmune disease
and can be triggered following a surgical infection, or by a flu-like
illness or a stomach infection. As the immune system fights off the
infection, it mistakenly attacks the peripheral nerves.

N-9 of the nurses' notes documents a precaution
for a "resistant bacteria" ,as evidenced by a check
mark in the upper right hand corner of that document, under the
subheading for "INFECTION CONTROL PRECAUTIONS". The
same precaution is also noted in the upper right hand corner of the
record at A-21. There are no further details.

At the time of her
admission to the hospital, Arlene Berry's blood pressure was
documented at "115/70 bpm, with a pulse of 79 and
regular", with signs of "mild diffuse weakness"
and "difficulty ambulating", evidenced at A-6. The
same record documents a respiratory rate of 18, on admission.
The normal adult respiration rate is 12 to 18 breaths per minute.
At the time of this assessment, Arlene Berry was found to be "alert
and oriented", with "NO Focal deficits".

The emergency department record at A-6, what I take
to be Dr. Spiller's physical examination, documents a "soft,
non-tender" abdomen, and "no
masses", suggestive of a typical
admitting physical note to express an overall, normal, negative
abdomen. A negative finding can also suggest hypotonia,
a disorder that causes low muscle tone that results in muscle
weakness. Constipation is
more common due to hypotonic gut musculature.
Hypotonia is often the
presenting sign for many systemic
diseases and diseases of the nervous
system. The abdominal muscles feel 'soft
and doughy'; also a sign of gastropareses
in clinical diabetes, which can rapidly
progress to intestinal obstruction.

On examination, the physician who saw her documented
positive bowel sounds, evidenced at A-6. Hyperactive bowel sounds
provide the most immediate indication of persistent upper GI
bleeding/GI hemorrhage. Gastrointestinal bleeding ALWAYS requires
prompt physician evaluation. An increased white blood cell count may
indicate an infection. Crampy abdominal pain can suggest acute
bleeding. Fatigue, shortness of breath, lethargy and pallor may also
be noted. Other findings may include hematemesis, coffee-ground
vomitus, bloody stools, Black or tarry stool, rectal passage of
bright red clots and jellylike material or melena. Decreased urine
output (urinary incontinence), tachycardia, and hypotension accompany
blood loss; can also suggest shock from excessive blood loss.

Arlene Berry was still neurologically responsive when
I saw her following her admission. She was able to reach and use for
herself the kidney basin at her bedside table, as she occasioned to
vomit more of the same flu-like "yellowish liquid"
that she had done so many times on the days before, and in fact used
it for herself in our presence, at which time a cool cloth was
provided by the nurses, as evidenced by the record at N-6.

The same record documents upwards of "100cc
yellowish fluid", what is frank bile, or 'bilious
vomit'. The time of that assessment was documented at 1915 hours on
May 23, 2000, following Arlene Berry's admission to the Kirkland and
District Hospital. The same record documents that the patient had
stated she was "very tired", whereupon she was then
assisted to bed, as evidenced at N-6.

Vomiting is a symptom of many causes. The clinical
difference between bilious and non-bilious vomiting (ie, vomiting
yellow or green) is critical in distinguishing life threatening
abnormalities.
The word "bilious" comes from the word
cholera. The word cholera is Latin for bilious disease and has come
to indicate a severe intestinal infection. People with bowel
obstructions may repeatedly vomit yellow, or green colored bile and a
history of frequent bilious vomiting in the presence of abdominal
pain should have been a 'red flag' suggesting intestinal
obstruction, which should have been treated emergently.

Arlene Berry also complained of being "cold"
and so the nurses provided her with extra blankets, evidenced at N-6.
Her very last words were that she was "feeling a little
better", also evidenced by that record.

A-26 documents a body temperature >37.0 .
According to the record at
the documented temperature is slightly above 37ºC at
approximately 37.8 suggesting a low-grade fever. Pathogenic bacteria
grow best at human body temperatures in the 37ºC range.

According to the medical record at N-6 Arlene Berry
was admitted at 18:45 hours and had spent 75 minutes in the ER, as
evidenced at A-3. In all that time, the ED physician, Dr. Spiller,
did very little. NO simple blood tests were done or even ordered at
that time. It is also clear that no course of action was charted,
marked by a clinically evident inability on the part of the ED
physician to adequately make a proper evaluation or even make a
provisional diagnosis. In fact, Dr. Spiller had no idea what to look
for, electing to play the "wait-and-see" game in the face
of life threatening indicators. Not only did the patient's family
physician fail to attend, NO diagnosis or differential diagnosis was
made following the patient's admission at that time, or at all. NO
protocols were followed.

A-3 of the record, what I take to be the physician's
diagnostic chart is a total blank. Again, from that record it seems
clear that nothing was entered because nothing was done. The same
record was filed out-of-sequence. The emergency record at A-4 was
also filed out-of-sequence. Interestingly both of these records were
dated using a rubber stamp that is consistent with backdating
techniques.

The record at A-12, what I take to be physician
orders documents a concomitant or concurrent administration of
Senokot (laxative), MS Contin (narcotic analgesic), Statex (morphine
family), and Gravol (an anticholingeric agent), including IV solution
and additives, the most dangerous of which is the MS Contin, a brand
name for "Morphine Sulfate".

Co-administration of narcotic analgesics such as MS
Contin with laxatives, ie. Senokot may have additive central nervous
system (CNS) and gastrointestinal (GI) system effects which can
increase the risk of severe constipation or paralytic ileus,
including CNS depression.

"Contin", is a
pharmaceutical industry buzzword for "time-release" or
"continuous" release. Additionally, Arlene Berry had been
given Statex (a narcotic: opioid agonist analgesic, also used to
relieve pain) which also belongs to a class of the morphine family.
Notably, morphine is contraindicated to sedation, brain tumors, or
increased pressure in the head or spinal cord, possible abdominal
problems requiring emergency surgery, in patients having a
substantially decreased respiratory reserve,

"MS" (morphine sulphate) is often
confused with 'Magnesium Sulphate'. Magnesium Sulfate is used to
treat severe constipation. Overuse of laxatives, or in combination
with bisphosphonates or Magnesium Sulfate, or an overdose of
magnesium sulphate used to treat severe constipation can lead to
hypermagnesemic pseudo-coma, which mimics a central brainstem
herniation syndrome.

According to the record at A-13, Arlene Berry was
given 30 mg (po bid) morphine by nurse McCrank at 2000 hours on May
23rd, the eve of her death in the face of an undiagnosed and
undifferentiated condition(s) associated with "abdominal pain".
Nurses do not dispense medications to patients without a doctor's
order. A judicious dose of morphine on standing
order to patients with non-traumatic abdominal pain is usually in the
range of 05. mg/kg.

The record at N-6 also documents telephone orders
received by the hospital from Dr. Jordan at 2030 hours for Stemetil
10mg by IV, 4 times daily for "control of nausea", given by
the RN, as further evidenced by the physician's orders seen at A-11.
Stemetil suppresses activity in the trigger zones of the vomiting
center by "paralyzing the gastrointestinal tract" which
governs the vomiting reflex, which can also exacerbate dismotility. A
typical single dose of Stemetil for a small woman with low body
weight is 5mg.

The antiemetic action of Stemetil (prochlorperazine)
may "mask the signs and symptoms of drug overdosage from other
drugs and may obscure the diagnosis and treatment of other
conditions". Increased sedation is a serious side effect of this
type of agent. Oversedation results in obtundation,
characterized primarily by reduced alertness and hypersomnia.

Hypersomnia is defined as a state of sleep in excess
of 25% of the expected normal. Further, phenothiazines have even been
reported to trigger diabetes in patients with no previous history of
diabetes.

Morphine and prochlorperazine
have a profound impact on bowel motility, often resulting in fecal
impaction.

The co-administration of a narcotic analgesic and
a neuroleptic agent will result in neuroleptanalgesia with
drug-induced reduction of oxygen intake, resulting in respiratory
depression. Respiratory depression represents the principal negative
variable introduced with "conscious sedation" and left
unrecognized and untreated, is the cause of panic, including most
serious complications.

Neurolept-analgesia, also called "conscious
sedation" refers to the use of major tranquilizers, ie
stemetil/prochlorperazine in conjunction with narcotics such as
morphine. Neurolept-analgesia is defined as a state of CNS
depression.

Notably, my wife had also been given penicillin
based medicines and sulfonamides such as Bactrim (Septra DS) and
CIPRO (cyproflaxin) on the days before her admission; penicillin and
sulfa-based antibiotics can cause the body's immune system to react
by over stimulation.
Septra DS is an antibacterial agent with a
wide spectrum of adversities (difficulty breathing; closing of the
throat; swelling of the lips; and unusual bleeding).

Cross-reactions between penicillins and sulfa-drugs
including sulfonamides are common triggers of drug-induced serum
sickness and fixed drug eruptions. Signs and symptoms of
overdosage reported with sulfonamides include anorexia, colic,
nausea, vomiting, dizziness, headache, fatigue, drowsiness, decreased
appetite. Hematuria may also be noted.

N-5 of the Nurses' Notes documents "Sudden large
queery bloody emesis,reddish brown liquid" at 0255 hours, on May
24th of 2000. Submit, when everything in the intestine slows down,
everything in it backs up.

N-3 documents an "Suctioned orally thick
brownish secretions"at 0320 hours (in the small hours of the
morning), suggestive of a more significant backup of intestinal
material, i.e., vomiting of fecal matter due to obstruction of the
bowel, evidenced by family present as "a large chocolate colored
(gross appearance), odorless, pasty material, looking pretty much
like feces". If you are unable to open your bowels due to an
obstruction somewhere, then your feces cannot exit your body via the
normal route and you can get nauseated and start to vomit fecal
matter. This condition requires urgent medical attention and probably
surgery.

The same record documents "suctioned down ET
tube several times for small amount of brownish mucous" (a
reddish brown liquid, suggestive of old blood or admixture of blood
and gastric content) at 0330 hours, while A-17 documents "being
suctioned for moderate amounts of coffee-ground emesis by RN" at
0330 hours on May 24th. Suctioning infers that the patient's airway
has become obstructed with secretions or debris. Any negligence of
the patient's throat secretions may lead to hypoxia, brain edema and
further deterioration in a patient's condition leading to a vicious
circle, which if not broken will lead to death.

The record at A-5 documents a blood pressure of
115/75 at 17:05 hours on May 23rd that by 18:45 hours had dropped to
100/50 bpm. Marked blood pressure lability with alterations between
hypertension and hypotension following paresis suggests an atypical
course of GBS.

A-20 of the record documents a Glucose
of 13.2 H mmol/L
(the normal range is 4.1 - 7.8). High blood sugar usually comes on
slowly. To convert mmol/l of glucose to mg/dl, multiply by 18. (13.2
x 18) = 237.6 mg/dl.

Random Blood Sugar Test (RBS) measures the level
of glucose in the body at any point of time. If the level of sugar is
between 140 mg/dL and 200 mg/dL, the patient is diagnosed with
pre-diabetes. If the level of sugar exceeds 200 mg/dL, then the
patient is diagnosed with diabetes. Glucose levels of 11.1 mmol/l
(200 mg/dl) at 2 hours confirms a diagnosis of diabetes. Symptoms of
severe high blood sugar include drowsiness and difficulty
waking up.

A-19 of the record documents an elevated WBC
Count of 22.4 H. WBC = Leukocytes. The
presence of an elevated WBC count is called Leukocytosis. White cell
count is actually 22,400. A normal WBC is 5,000 to 10,000. Normal
Adult Range: 3.8-10.8 thous/mcl Optimal Adult Reading: 7.3
WBC leukocytes are the body's primary defense against bacterial
infection and also reflect the degree of physiologic stress. WBC's
are also elevated with dehydration, and hyperviscosity secondary to
dehydration. If the total WBC is high due to a
rise in neutrophils and eosinophils, then an allergic,
or parasitic process
is most likely. An increase in the WBC count (leukocytosis) is also a
typical response to noxious stimuli.

The record at A-19 documents a Neutrophil count
of 92.0 H with an absolute neuts of 20.0 H.
Neutrophils, are also
known as "segs","PMNs"or
"polys"(polymorphonuclears). CSF in bacterial meningitis is
typically dominated by the presence of PMNs. PMN’s generally
predominate in bacterial infections. Patients with subdural hematoma,
metabolic abnormalities, or meningitis may seem to be
encephalopathic.

"The presence of
polymorphonuclear granulocytes does not rule out the diagnosis of
Guillain-Barré syndrome". Eur J Neurol 10(5):
479-86.

Neutrophilia (or neutrophil leukocytosis)
is a condition where a person has a high number of neutrophil
granulocytes in their blood. Neutrophilia may be due to a number of
acute and chronic causes such as infection, inflammation, emotional
stimuli, drugs, metabolic hormonal, and endocrine disturbances,
including hematologic abnormalities. Wegener's granulomatosis,
granulomatous cerebral amebiasis, vasculitis, and heart attack are
high on the order of Neutrophilia.

Leukocytosis, especially neutrophilia, indicates
systemic infection and is rare in the absence of bacterial
"superinfection", also called "superbugs" are
bacteria, viruses or mixed infection which are resistant to
antibiotics.

The record at A-19 documents a Lymphocyte
count of 2.0 L(low) suggestive of lymphocytopenia in which
lymphocytes (T-cells) are reduced with nutritional deficiency,
infection, and ascites due to "fluid build-up in the abdomen",
and/or an exhausted immune system. If bacterial infection is present
in ascites this may suggest spontaneous bacterial peritonitis in
which abdominal pain is a prominent finding. If peritonitis is not
treated promptly and effectively multi-system organ failure occurs
rapidly.

A-19 documents a Red Blood Cell (RBC) Count of
4.30(3.80 - 5.80 is normal), but the HCT (Hematocrit) is very low,
with a reduction suggestive of anemia. Anemia is also a prominent
cause of dyspnea when the hemoglobin concentration falls below 8-10
g/dl.

The same record seen at A-19 documents an HCT
count of only 0.361 L (low): HCT is the measurement of the percentage
of red blood cells (RBC's) in whole blood. The hematocrit (HCT) is
another way of measuring the amount of hemoglobin (Hb), and in this
case it is very low. Thus anemia is present when HCT is <>1.0 x
109/L.

The monocytes are a type of phagocyte which
mature into "macrophages"; they are important germ eating
cells. The majority of patients with Guillain-Barré syndrome
will have 10 or fewer monocytes.

Patients with a low monocyte count have a higher
risk of getting sick from an infection, particularly those caused by
bacteria. In cancer, or leukemia, the monocytes become elevated. In
this case the monocyte count is well below the normal range.

A-20
documents an O2SAT (oxygen saturation)- arterial oxygen saturation
(SaO2) of 98.9 H, with with a NORMAL reference and an evident run
time of 1720 hours, notably several hours after the patient's alleged
time of death, following her transfer out to Sudbury on May 24th of
2000.

The same record documents an Arterial Ph of
7.437, in the normal range. The time of that assessment is
documented at 0400 hours. Hydrogen ion concentration expressed as pH
"Power of Hydrogen". A Normal pH is 7.35 - 7.45.
Neutral pH is 7. For example, the pH of blood is normally 7.4 and
that of muscle is 7.0. pH under 7 is acid; pH over 7 is basic or
alkaline. The metabolic pathways of the body require a slightly
alkaline environment. Anything below 7.20 is generally
considered critical. At 7.0 the heart will stop beating.

A-18 of the medical record documents an "inferior
ischemia", a sign of reduced oxygen supply to vital organs due
to reduced or poor blood flow to the heart. An "inferior
ischemia" is the hallmark of "impaired organ perfusion",
as it implies that, unless corrected, there may not be enough oxygen
in the blood to sustain vital organs.

The same record at A-18 documents "Sinus
Tachycardia". Sinus tachycardia occurs when the sinus rhythm is
faster than 100 beats per minute. The rhythm is similar to normal
sinus rhythm with the exception that the RR interval is shorter, less
than 0.6 seconds. P waves are present and regular and each P-wave is
followed by a QRS complex in a ratio of 1:1. At very rapid rates, the
P-waves might become superimposed on the preceding T waves such that
the P waves are obscured by T waves. Sinus tachycardia, (>90/min),
is seen in over 35% of patients with Guillain-Barré Syndrome,
and over 30% suffer from hypertension (Parry, 1993).

A-20
documents a Sodium level of 144 (137 - 145) mmo1/L. Sodium is
an electrolyte that helps with nerve and muscle function, and also
helps to maintain blood pressure. Sodium circulates in the body
fluids outside the cells. It is very important for maintaining blood
pressure. Sodium is also needed for nerves and muscles to work
properly.

CAVEAT: Hyperglycemia can lower
the serum sodium concentration by 1.6 mEq/L for each 100 mg/dl, also
giving rise to a false test.

A-20 also documents a serum potassium level of
3.4 L at 0400 hours on May 24th of 2000. Low potassium is defined as
a potassium level below 3.5 mEq/L.

A-20 of the hospital record documents a CK
(Creatine Kinase) level of only 40 units per liter (U/L) at 0400
hours. CK is the most sensitive enzyme and in the presence of most
diseases, levels can be elevated as much as 50 to 100
times the reference level. In females, total CK should typically
be 10 to 79 units per liter (U/L).

Metastatic malignant neoplasms
cause severe body wasting-cachexia. The hallmark of muscle
damage or muscle wasting is elevation of CK concentration. The
wasting away of fat and muscle (cachexia) is the most visible
hallmark of metastatic cancer. Persons with cancer
typically have high CK levels.

Notably, Guillain-Barré syndrome does not
produce evidence of muscle inflammation such as elevation of the CK.
Further, a normal CK (CREATINE KINASE) at the time of the patient's
admission would argue favorably against a diagnosis of metastatic CA,
at that time.

EVIDENCE OF SUBSTANDARD CARE

N-10 of the Nurses' Notes document the patient's
level of care as "routine", which showed little or NO
concern for patient safety. Further, NO close patient monitoring or
toxicological screening was done, marked by a complete absence of
nursing care plan, as evidenced at A-21 of the medical record. In
fact, NO inherent blood work was done in a timely manner. NO
protocals were ever followed or implemented, in this
case.

Stemetil is widely distributed into body tissues
and fluids. Stemetil undergoes metabolism in the gastric mucosa and
on first pass through the liver where it enters the entero-hepatic
circulation and is excreted chiefly in the feces.

Stemetil can also lead to changes in the blood-brain
barrier (BBB), allowing an infectious agent to gain entry to the
brain and produce lethal central nervous system (CNS = brain and
spinal cord) infection. The scientific literature describe two
bacterial factors specific to the meningitis pathogen that thwart the
normal protective role of the blood-brain barrier, leading to serious
infection.

Further, sugar solution in IV creates gaps in the
blood-brain barrier allowing chemicals to enter. Infected material
can block the blood vessels to the brain, and Stemetil can help
shuttle it directly into the brain and CNS. Once across the
blood-brain barrier, the infection enters neural cells, with
resultant disruption in cell functioning, perivascular congestion,
hemorrhage, and inflammatory response diffusely affecting gray matter
disproportionately to white matter.

Blood borne infection in the blood lyse easily.
It seems logical to assume that Stemetil would be contraindicated to
serious infection for this reason.

Stemetil poisoning is marked by
oversedation, respiratory depression and hypotension. Stemetil
(prochlorperazine) intoxication or poisoning can also cause deep
physiologic depression that resembles and can mimic brain death

It is also clear that Dr. Jordan sought to
eliminate the symptom "nausea", without his attendance, as
evidenced by the phone order"for control of nausea" and
without any appropriate blood testing, or addressing any possible
underlying causes.

Further, Dr. Jordan neglected to consider the
etiology of the nausea and vomiting as a condition requiring prompt
medical intervention. Instead, he elected to give the patient a brain
damaging neuroleptic antipsychotic-antiemetic drug without any review
of her medical record, and without the benefit of toxicological
screening or close monitoring, evidenced at A-21. Clearly, the
etiology of the nausea and vomiting had never been determined, as
evidenced at A-3.

The record at 0020 hours seen at N-6
documents the discovery by duty nurses of the patient's "head
against the left side bed rail with her feet under the right side
rail". Sensory loss in GBS, if present, takes the form of
proprioception (loss of sense of one's own perception of the relative
position of neighboring parts of the body to each other), which is
occasionally impaired spontaneously, especially with extreme fatigue.

The ED physician, Dr. Mark Spiller was up to
assess the patient's condition. Upon examination her eyes were
documented as being "sluggish". She was simply repositioned
by the nurses, as evidenced by the record at N-6. Her"pupils
were dilated at approx. 5 mm" with "very little reaction to
light", and far from getting better she was becoming
progressively worse, as evidenced by a sense of urgency seen on the
record to the attendance of the patient with increased activity
evidenced at N-6 between 0030 and 0055 hours, also noted at N-5.
Clearly, from that record and apart from running around the room
looking busy and repositioning the patient, nothing was done. It
seems clear that the ED physician failed to properly assess the
patient's condition, which fell far below an acceptable standard of
care. Further, to add insult to injury, NO blood-work had yet been
done.

While the clinical feature of 'fixed dilated pupils'
is a valuable clinical sign it does not necessarily mean that the
patient has severe brain injury.

I assume that Dr. Jordan would have been alerted
by phone. He claims to have called in at 0100 hours but nevertheless
opted not to change his orders, as evidenced by the "no change
in orders" seen at N-5. From that record it is clear that Dr.
Jordan elected to alienate and treat the patient over the telephone,
unseen, in the face of life threatening indicators, all of them
ignored and without ever having reviewed the patient record.

Further, between 0200 hours and 0220 hours the
patient's blood pressure had risen slightly from 150/72to 162/80, a
sign of mounting hypertension such as caused or worsened in response
to treatment. The record at A-26documents the time of that assessment
as 0220 hours, while N-5 documents the time of the same assessment at
0230 hours, a 10 minute difference. The same record documents a HR
(heart rate) in the 160's, what is termed "sinus
tachycardia".

A-26 documents a blood pressure of 162/80 with an
SaO2 of 80% at 0220 hours, followed by a lethal drop in blood
pressure to 78/70 by 0235 hours, in which blood pressure rises or
falls significantly, a hallmark feature of alternating hypotension
and hypertension.

CAVEAT: Systolic blood pressure <80
mm HG is a hallmark of haemodynamic instability. The term
"hemodynamic instability" is most commonly associated with
an abnormal or unstable blood pressure, especially hypotension, or
trauma due to clinical insult.

The record at A-17 documents a complete cessation of
the use of abdominal and accessory muscles, evidenced by a "0
use of acc muscles"; and a "0
use of abd muscles"; the muscles of
respiration, accessory muscles and diaphragm are affected,
suggestive of respiratory and accessory muscle paralysis (crisis)
requiring intubation and mechanical ventilation. Functional
abdominal muscles also play a role of in conjunction with the
accessory expiratory muscles. The time of that
assessment is documented at 0330 hours.

Accessory muscle paralysis will
result in apprehension and anxiety.

EVIDENCE OF ALTERED RECORDS

There are numerous material deficiencies in the
related medical record of Arlene Berry which manifest a complete lack
of internal consistency, ranging from out of sequence records, from
the physician's Discharge note seen at A-1 and A-2, which is mared by
error, inconsistency, omission, and contradiction, to the nurses
Triage, to obviously rewritten, altered, and falsified medical
records, tailored to obfuscate the truth, seen between N-1and N-3 of
the nurses notes, with A-16 and A-17 presenting similarly, including
as follows:

A-26 of the record documents a BP (blood
pressure) of 78/70 at 0235 hours, while N-5 documents a BP of 98/70
at the very same time, suggestive of copious error.

A-24 documents a heart rate of 174 bpm at 0330
that is consistent with "trauma", while the Ventilation
Record documents a heart rate of only 126 at the very same time, a
significant difference.

A-4 of the record, what I take to be a Trauma
Legend, barely visible in the physician’s notes situated in the
lower right hand side of that record, there is an "obliterated"
area suggesting a white-out, or perhaps an erasure. From that record
it seems clear that relevant information was deliberately withheld,
or removed to conceal an event.

TRAUMA is defined as
any insult to the body, clinical or otherwise.

The record at A-6 documents a "history of
metastatic lung cancer", while the outpatient record at OP-54
clearly documents "no metastasis"and "mediastinoscopy
negative".

N-4 and N-5 present with less than half a page of
documentation consistent with deliberate omission, such as having
rewritten that record for the express purpose of withholding
incriminating information.

A-16 documents a blood pressure of 163/117 at
0330 hours, while N-3 documents a blood pressure of 136/85 at the
very same time. The same record documents a blood pressure of 121/81
at 0400 hours, while N-2 documents a blood pressure of 112/57 at the
very same time. More copious error.

N-4 of the record documents that Dr. Jordan was
called in at 0225 hours. A-1 of the record documents "I was
called in later that night because the patient had become obtunded",
while the record at N-2 documents "attempts to pull away to
painful stimuli" as late as 0400 hours on May 24th, being one
hour and thirty-five minutes later, according to the record.

Was it the doctor's belief that Arlene Berry
ceased to be a human being after becoming unresponsive following
undiagnosed, untreated and/or inappropriately treated conditions? So
much so that he decided to write her off?

N-10 of the record which documents the patient's
bowel routine and urinary elimination pattern for toileting is a
complete blank, with the very same information that ought to have
been recorded, also omitted at OP-53 of the record.

The record at N-5 documents a physician "assessments
unchanged" despite the fact that the patient had already gone
into respiratory distress, as evidenced by "Cheyne-Stokes"
respirations with periods of"apnea"lasting"5-8
seconds".

Sleep Apnea means"cessation of breath".It
is characterized by repetitive episodes of upper airway obstruction
that occur during sleep, usually associated with a reduction in blood
oxygen saturation. Other causes include panic attacks.

Low oxygen saturation may be present with advanced
respiratory muscle involvement. If proper balance is not restored or
corrected, the heart and lungs may fail and the brain will literally
begin to suffocate.

The record at N-5 documents the respirations as "deep
and soaring without constant jaw lift"as early as 0220 hours.
A-26 of the record documents "gurgling",and "snoring"
and is evidenced in the lower left corner of that record.

N-5 of the record documents "family in" at
0250 hours. On seeing the patient, we found her to be propped up in
the arms of two nurses, gasping for air, with only a plastic oral
airway in her mouth. A reason for this , according to the duty nurse
was "to keep the patient from swallowing her tongue".
It is also of interest to note that NO attempt was made
by either of the doctors to place the patient in the ICU in a timely
manner.

Weakness of tongue and retropharyngeal muscles causes
positional airway obstruction; difficulty with protruding tongue and
difficulty swallowing indicate that bulbar involvement is
significant, which requires that these patients be placed on
ventilators in order to breathe. Snoring, sleep apnea and gasping for
breath are part of the same problem. Failure to manage the airway
with endotracheal intubation when necessary or in a timely manner is
clear evidence of negligence.

I had asked the patient twice, in the presence of her
foster brother, if she could hear me to wiggle her toes, and indeed
she did, not once but twice, to be absolutely certain. An observation
made by her foster brother as he gently stroked her right cheek was
the seeming appearance of the patient attempting to pull her face
forward as though trying to lift her head off the pillow. The
inability to lift the head off the pillow by flexing the neck is
another danger sign associated with GBS; it frequently develops
simultaneously with phrenic nerve (diaphram) weakness.

In my opinion, Arlene Berry appeared to be more
paralyzed or blunted than anything, with the exception of lower limb
joint contractions which rapidly diminished and became hyporeflexic.

Her condition was cataleptic-like, characterized by a
profound hypnotic state, or psychomotor condition of morbid sleep,
such as seen in cataplexy, neurolepsis, sleep paralysis, or
narcolepsy. Underlying causes of catalepsy include severe emotional
trauma, and emotional shock. Compare: neurolept-analgesia (conscious
sedation)in combination with a severely paralyzed motor function.

Although patients with GBS in the setting of
preserved consciousness may appear to be completely obtunded, they
are technically awake and fully lucid. But he/she may literally not
be able to move a muscle in response. The GBS patient only appears to
be unresponsive due to a severely paralysed motor function. It has
been shown that more than half of the time it is the family and not
the physician who first realized that the patient was aware.

With GBS the patient is conscious but unable to
respond due to a severely paralysed motor function.

N-3 of the record documents "resp
noisy","shallow","Cheyne-stoke"at 0320
hours. Cheyne-stokes breathing is a respiratory pattern that
oscillates between hypoventilation and hyperventilation, usually the
result of diencephalic insult. It is also seen during sleep in some
normal individuals.

Noise heard during any part of the respiratory cycle
may indicate airway obstruction or alteration in airway patency.
Patients with neuromuscular disorders have rapid, shallow breathing
secondary to severe muscle weakness, which requires that these
patients be placed on ventilators in order to breathe.

The record at 0255 hours documents a"sudden
large bloody-emesis of reddish brown"or what is known in medical
circles as "coffee-ground emesis" ie. dark brown tinged
vomit the color and consistency of coffee-grounds, composed of
gastric juices and old blood, which can rapidly grow bacteria.

Vomit that contains blood may have a red or brownish
appearance and is called coffee ground vomiting indicating that it
has come from large intestines, suggestive of a slow bleeding source
in the upper GI tract. Obstruction below the middle of the small
bowel also gives rise to brownish vomit.

Gastrointestinal (GI) bleeding due to stress
ulceration in GBS is reported in the literature.

GI bleeding is considered a potential medical
emergency. It involves assessing hemodynamic stability, resuscitating
the patient as needed, locating the source of the bleed, and treating
the underlying cause.

From the record it seems clean that NO emergency
measures were taken with respect to GI bleeding and that this medical
EMERGENCY event was met by the doctors with complete indifference.

The record at N-4 documents "incontinent blood
tinged urine"at 0305 hours. Incontinent "tinged urine"
is consistent with severe dehydration, often mistaken for hematuria
(blood in urine). Incontinence can be the result of hypotonia, or
neurogenic bladder.

Notably, N-3 of the record documents a "large
amount of dilute urine" (polyuria) at 0325 hours, only 20
minutes later. This finding would be inconsistent with hematuria.

The record at N-2 documents "Foley draining lge
amt dilute urine" at 0425 hours, while N-1 of the record
documents "Foley catheter emptied for 1200cc dilute urine"
at 0450 hours that is consistent with conditions featuring osmotic
diuresis and by diabetes insipidus ("water diabetes") It
occurs in association with Na+ Disorders, primarily related to Na
negligence due to iatrogenic fluid overload.

Na is lowered 1.6meq/L for every 100mg/dl glucose.
The central causative mechanism in this case, involves a
hyperglycemia-induced osmotic diuresis and resultant dehydration.
Polyuria due to excess fluid intake and glucose-induced osmotic
diuresis is common in patients with transient hyperglycemia. The
hyperglycemia emanates from a commonly identified diabetogenic
stressor, such as infection, which precipitates the onset of the
syndrome, which in turn produces pseudohyponatremia commonly
associated with hyperglycemia.

A-15 documents the 24 hour IV fluid balance record,
that between 1745 hours and 0200 hours was administered as follows:

A-14 documents an " IV gid prn", meaning
that fluid and medication rate of administration to be is given by IV
as follows: "2/3 and 1/3", being a 3.3 % dextrose and 0.3 %
sodium chloride @ the rate of 100 cc/hr, as evidenced at A-15.

The same record documents a total Na of 1000 cc TBA,
(to be absorbed), documented by nurse Bates at 1745 hours; with a 150
ABS (absorbed) by 1900 hours, documented by nurse Ferguson, shows 150
cc absorbed over a period of 75 minutes; far exceeds the rate of 100
cc/hr. Anything above 100 cc/hr points to "overly rapid
infusion".

Na 850 cc TBA remaining at 1900 hours documented as
ABS (absorbed) by 0200 hours, over a period of 7 hours, resulting in
a "surplus" of 150 cc fluid, with an additional 1000 cc
TBA, and no further documentation with respect to Na monitoring. The
ventillation record at A-16shows a complete absence of information
with respect to "Water Refill".

Hyponatremia results from a "surplus of water"
due to Na negligence. Other evidence of fluid overload as indicated
by "ascites" due to fluid build-up in the abdomen.

Rapid correction of hyponatremia, even mild
hyponatremia, risks neurologic complications (see Fluid and
Electrolyte Metabolism:Osmotic demyelination syndrome). Generally, Na
should be corrected no faster than 0.5 mEq/L/h. Increase should not
exceed 10 mEq/L over the first 24 h. Any identified cause of
hyponatremia is treated concurrently.

The record at N-6 documents "IV infusing
well".There are no further IV related entries on that record, or
any other record, either to indicate when or if the IV was
discontinued, or to show that the rate of administration was being
monitored, suggestive of patient dumping, or abandonment, with
deliberate omission by reason of Na negligence.

The same record documents the use of a No. 20 "Quick
Catheter"; signed by nurses Bates, Ferguson, and
McCrank (may or may not be relevant). RN is ultimately
responsible for monitoring rate of infusion.

The same record documents a "hard" IV site
in the "R" (right) hand; clot formation due to irritation,
of the vein from solution or medications is the most common cause of
a hard IV site. The back of the hand has weaker veins, and is not
commonly used for IV antibiotics. Seriously ill patients require
accurate fluid balance monitoring because IV fluid also contains the
medication(s). Rapid infusion may also lead to overdosage.

Circulatory overload can occur if IV is not regulated
properly and IV fluids infuse too rapidly for the patient's body to
handle. Signs of fluid overload include tachycardia, elevated blood
pressure, dyspnea and other signs of respiratory distress.
Neuromuscular disease is another well-known cause of dyspnea.

Notably, Stemetil 10mg was added to the IV at 2030
hours. The drug is sedating and a potent vasodilator, which also
crosses the blood-brain barrier. Patients are usually "volume
expanded" prior to its use, often due to negligence, resulting
in neurologic derangement.

Hyponatremia is diagnosed by measuring serum
electrolytes. However, serum Na may be artifactually low when severe
hyperglycemia increases osmolality. Water moves out of cells into the
ECF. Serum Na concentration falls about 1.6 mEq/L for every 100-mg/dL
(5.55-mmol/L) rise in the plasma glucose level above normal. This
condition is called translational hyponatremia because no net change
in the amount of Na has occurred.

The record at N-4 documents the patient's "transfer
to ICU in respiratory distress" (sudden breathlessness) at 0320
hours, while record at N-3 documents a "congested oral airway"
at the very same time, meaning congestion of the breathing passages.
Obstruction of the air passages of the nose, mouth, or throat may
also lead to difficulty breathing.

Certainly the inability to breathe properly can be
alarming, and many persons will immediately react with anxiety, fear,
or panic.

The record at A-24 documents the mechanical charting
of the patient's vital signs that commenced recording at 0315 hours.
It is interesting to note that the patient's transfer to the ICU had
not yet taken place, and that no prior attempt was made by any of the
healthcare providers to place the patient in the ICU prior to that
time. It seems clear that the healthcare provider had done too little
too late as evidenced by the records at N-9, N-10, N-11, including
A-3, and A-21.

From the record as a whole, it is also clear that
both doctors should have realized at the onset, from the severety of
the patient's signs and symptoms that they were faced with a
critically ill young woman who was not responding to their
questionable treatment. They should ALSO have been acutely aware of
the danger.

Critically ill patients frequently have multiple
physiologic derangements that come from a range of possible sources
and occur simultaneously.

N-5 of the medical record documents a gurgly
respirations as evidenced by a "gurgly resps" at 0220
hours, a sign of constriction suggestive of thoracic trauma (patients
are often in shock). The same record documents "deep and soaring
and without constant jaw thrust", such as associated with the
airway and swallowing difficulty in respiratory compromise. Gurgling
is a bubbling sound. It usually indicates upper airway obstruction
from secretions, emesis, or blood. Gurgling respirations indicate the
presence of fluid in the airway, usually blood or vomit, or both.

The record at N-4 of the Nurses' Notes documents
"incontinent blood tinged urine"at 0305 hours that is
consistent with urinary incontinence (leakage of urine) or blood
tinged urine if bladder infection is also present. Urinary
dysfunction such as incontinence (leaking) of urine is a prominant
finding in GBS patients due to autonomic abnormalities. Incontinence
is loss of bladder control, and is also a very serious side effect of
antipsychotic medications. Further, during episodes of hypokalemic
periodic paralysis urinary output is decreased during the attack
because water accumulates intracellularly in muscles.

Dark, concentrated urine in decreasing amounts
(incontinent tinged urine)is also a prominant finding in patients
with high blood sugar, including dehydration.

A-8 of the related record documents "patient was
unconscious with respirations of approximately 30 and labored",
that is consistent with dyspnea- difficult or labored breathing.
Dyspnea is breathlessness due to high filling pressures and pulmonary
congestion/edema, i.e. shortness of breath, a smothering feeling,
inability to get enough air, and suffocation. Breathing may become
labored and difficult; labored breathing is the hallmark of
respiratory distress and respiratory failure due to paralysis of the
diaphragm. Dyspnea should always be taken seriously.

A-1 documents "plantars upgoing bilaterally".
Submit that the plantar reflex is a hallmark of the Babinski sign, a
test for signs of disease process in the `motor neurons` of the
pyramidal tract. Initial drowsiness, bilateral plantar responses, and
quadriparesis, is strong clinical evidence of central involvement
consistent with drugs or toxins that affect the basal ganglia,
thalmus or brain stem. Babinski's sign is also a prominent finding in
Bickerstaff's brainstem encephalitis (BBE), a variant of the Guillain
Barre syndrome. Further, "limb weakness in GBS is nearly always
bilateral" - (Parry, 1993).

The lower limb is often ischemic in diabetes. Fecal
impaction as a cause of acute lower limb ischemia is also reported in
PubMed.

The patient became apparently unresponsive, as
evidenced at N-5, and went into respiratory distress, requiring
ventilation for which she was transferred into ICU at 0320 hours,
according to the record at N-3. The same record documents the time of
the patient's intubation by Dr. Jordan at 0325 hours, 5 minutes
later. What I take to be the Ventilation Record at A-17 documents the
arrival in the ICU of the hospital's ventilatory therapist, Helene
Studholme at 0330 hours, after being "called in for patient
requiring ventilation". From these records, it is clear that
either the ventilatory therapist was not present at the time of the
intubation procedure because she did not show up until 5 minutes
later, or that the intubation did in fact take place at an earlier
time, such as 0320.

Notably, the record at A-24 documents a HR (heart
rate) of 174 bpm at 0320 hours that is consistent with an "awake
intubation", (any suspicion of difficulty intubating, for any
reason), marked by panic with "awareness".

To illustrate, the Vital Signs Record at A-24
documents a heart rate of 174 bpm at 0330 that is consistent with
"trauma", while the Ventilation Record seen at A-16
documents a heart rate of only 126 at the very same time, a
significant difference, suggesting that the time line for that event
was in fact altered by the Ventilatory Therapist to obfuscate
iatrogenic trauma related injury. The Vital Signs Record is a
mechanical record with a run time, while the Ventilation Record is a
handwritten account, marred by having been rewritten. Which is more
likely to make copious errors or downplay an event by omission or
telling lies?

There is nothing on record to suggest that anesthesia
was or wasn't given to prepare the patient for the intubation
procedure. My opinion is that if its not on record it didn't happen.

The earliest indication of shock is an increase in
heart rate (HR).

According to Dr. Jordan "the intubation
proceeded uneventfully", while N-2 of the record documents the
ET (endotrachial tube) "pulled back 4 cm" at 0425
hours. From that record it seems clear that the endotrachial tube had
been malpositioned for almost one full hour before the error was
discovered by one of the nurses, as evidenced by the record at N-2;
infers negligence on the part of the Dr. Jordan, including
failure on his part to identify an incorrectly placed airway in
a timely manner. Both myself and the patient's foster brother were
present to witness that event.

Mal positioning of ET tube can cause airway
obstruction and may also result in tissue trauma, and bleeding. When
an endotrachial tube is misplaced in the esophagus and misplacement
is detected late, a compromise of the patient's safety can be
significant.

A-12 of the medical record documents a blood pressure
of 163/117 bpm at 03:20 hours that by 03:45 hours had dropped to
85/58, following intubation, with an additional drop to 85/52
bpm by 3:52 hours, over a span of some 7 minutes, as evidenced at N-2
in the Nurses' Notes.

A-17 documents "being suctioned for moderate
amounts of coffee-ground emesis by RN" at 0330 hours.
Gastrointestinal bleeding due to stress ulceration is also an
important complication in critically ill patients. GI bleeding is a
medical emergency that was basically ignored by the health care
providers, in this case.

Stupor and coma are characterized by impairment of
the arousal system. In stupor, a person arouses only in response to
strong verbalor tactile stimuli, awakens briefly, and then lapses
back into a sleeplike state after the stimulation stops. In coma, a
person cannot be roused to consciousness. With GBS the patient is
conscious but unable to respond due to a severely paralysed motor
function.

Besides dangerous cardiac manifestations,
neuro-endocrine changes are also reported and could induce
electrolytes and fluid balance impairments. Polyuria has been
observed in a severe case of GBS. Polyuria in GBS is multi factorial
and would be partly due to a dysregulation of osmo-receptors.

The physician's Lab Work Summary at A-19 documents
the charting of a course of Hematology (blood-work) and Coagulation.
The same record documents a Fibrinogen level of 4.67 H (the normal
range is 2.00-4.00). Elevated fibrinogen levels induce a state of
hypercoagulability.

When blood protein is high, CSF usually
clots because of the presence of increased fibrinogen. Serum
fibrinogen levels in a safe range is <300 mg/dL. The plasma
fibrinogen level appears to reflect disease activity in acute
Guillain-Barré syndrome and is typically elevated at
presentation. In fact, ongoing activity of Guillain-Barré
syndrome may be reflected by a persistently elevated fibrinogen
level. Acute phase reactant: marker of inflammation Further, elevated
levels may also be seen with TRAUMA of any kind.

A-19
documents a D-dimer test level of 1000 H (<500) fibrinogen
and d-dimer correlates
with thrombotic activity)
suggests thrombosis. Thrombosis signifies the formation of blood
clotting within vessels of the brain or neck.
People who are suffering from a severe infection are more likely to
develop dangerous blood clots,
but inappropriate combinations of medications or treatment can
sometimes be the worst offenders.

The Cardiac Index at A-18
documents the patient's ventilation rate at 129 bpm (breaths per
minute) at 0417 hours, with heart and breath rate increased.
Increased heart and breath rate can suggest clinical insult, such as
caused or worsened by medications, resulting in oxygen deprivation.
The Cardiac Index documents the patient’s age at "55 years",
she was only 41 at the time of her death; can imply negligence, or
even patient record swapping.

A-19 documents the aPTT=
activated Partial Thromboplastin Time, a test used to determine the
efficacy of various clotting factors used in the diagnosis of
coagulation disorders documents the therapeutic range for heparin
therapy at 60-100 seconds (23-35 is the normal). The time of
that assessment was documented at 0400 hours. The aPTT is typically
elevated in 90% of those with coagulopathy.

A-19
documents a "PLT ESTIMATE" - "MOD INCREASE"
confirming a moderate increase in platelet
aggregation activity (blood platelets sticking together), indicating
that blood thinners may be needed to prevent blood clots.

The
ambulance call report seen at N-7, of the Nurses' Notes documents
that the patient was intubated and vented and that she was seen to be
"stable", but that she appeared to be "pale, dry and
cool". In patients with GBS, the skin may become pale and dry,
and sweating may become reduced. Body temperature can also be marred
by the effects of drug-induced temperature dysregulation, which can
suppress sweating, causing central nervous system impairment, often
resulting in an afebrile state. Cool, dry skin can also suggest late
sepsis, a prominent finding with iatrogenic neglect.

Hemodynamic
instability has also been defined more broadly as global or regional
perfusion that is not adequate to support normal organ function. This
definition recognizes the obligation to insure adequate organ
perfusion during the flaccid period in patients with GBS.

According
to the Nurses' Notes at N-1 of the record the patient was given
Gravol 50 mg x 10by paramedics at 0620 hours, while the record at
N-7with respect to medications documents "See Nsg Notes".
Gravol (dimenhydrinate) is contraindicated in lung disease and has
also been reported to "mask the presence of underlying organic
abnormalities and/or the toxic effects of other drugs".

The
record at A-8 and A-9 documents "Medi-Vac team were due to
arrive at 0435", while the Ambulance Call Sheet documents "call
received at 0620" hours, a significant difference. The
record at N-7, documents "pulses X 4 good"; head and neck
OK; chest OK; "abdomen OK"; pelvis OK; extremities OK.

Following her transfer to Sudbury on May
24th of 2000, Arlene Berry's remains was returned to Kirkland Lake
several days after family had been notified of her death via her
foster brother acting as a family contact, who was notified by phone.
On seeing the deceased victim, her eyes were "sunken in
appearance", with swelling and distortion of the face, eyes, and
lips, as was the case, marked by a rash-like redness resembling a
sunburn with "blistering" wrinkles in the skin in the area
just below the right eye, consistent with a delayed hypersensitivity
reaction, or fixed drug-eruption, evidenced by all who attended
Arlene Berry's viewing at the Monette Funeral Chapel in Kirkland
Lake.

It is believed that Arlene Berry's death was
deliberately provoked and that her eyes were taken by Drs. Sauvé,
and Adegbite at the Sudbury Regional Hospital, upon remote third
party consent (foster brother), utilizing deception to obtain that
consent, by-passing permission from Arlene Berry's immediate family,
ie, her de facto common-law spouse and her children. Only biased
clinicians might provide less aggressive scrutiny, withhold medical
treatment and influence the family in inappropriate ways such as
this. Indeed, the fraudulent taking of the patient's eyes in the
manner in which it was done can only be construed as theft. From the
information at hand, it seems clear that Drs. Sauve and
Adegbite sought to open the way, under misleading conditions
(influence of drugs, and metabolic disturbances) to organ donation
from brain death. The diagnosis of brain death allows organ donation
or withdrawal of life support. These doctors allowed this patient to
die to achieve their nefarious ends. Certifying brain death to
cover-up medical blunders or to increase organ donations constitutes
murder.

At a first meeting with the coroner held at the OPP
Detachment in Kirkland Lake sometime in July of 2001, Dr. Barry A.
McLellan, who was the Regional Supervising Coroner for northeastern
Ontario at the time admitted to family that there was "no
evidence on record to suggest matastasis", meaning spread of
cancer. In fact, there is nothing at all on this record to support a
diagnosis of cancer in the first place. For the record, fungal,
mycobacterial, parasitic, and indolent bacterial infections have been
known to occasionally mimic cancer. In fact, certain pulmonary
infections can mimic pulmonary neoplasms.

At a subsequent
meeting between family and Dr. McLellan, he provided us with a view
of a CT scan that was purportedly done in Sudbury, Ontario at the
time Arlene's death on May 24thy of 2000, although I suspect it may
have been done following withdrawal of life support. It shows
multiple lesions of undetermined origin. The pathogenesis of these
yet undetermined lesions remains unclear but a metabolic disorder
seems the most plausible pathological factor. Based on the patient's
belated CBC's, the lesions are consistent with collections of
purulent exudates (pus producing bacteria), suggested by an elevated
Neutrophil count, and/or pockets of pooled blood, as suggested by an
elevated Fibrinogen in the presence of an elevated D-dimer, a
hallmark of thrombus formation, ie., blood clots. The enhancement is
obviously due to infection, or blood pooling, or both.

A-1 of
the record also documents "she died several days later with
numerous metastatic lesions to her brain". According to her
death certificate, Arlene Berry died May 24th of 2000, the very same
day she was transferred out to Sudbury. As to the cause of death,
according to a Dr. Sauve in Sudbury, she died "meeting brain
death criteria".

No attempt to diagnose was made at that
time, or at all. No pathological reason was given for the declaration
of brain death. No process of exclusion was undertaken without which
a diagnosis of brain-death should never have been considered.

No
autopsy was performed. No appropriate period of observation and/or
trial of therapy was ever undertaken. In fact, Arlene Berry was
rushed to her death within five and one-half hours of her departure
from Kirkland Lake to Sudbury, some 210 miles away. One might ask how
much time did these medical dolts actually spend assessing the
patient before pulling the plug on her?

Although
many conditions can mimic brain death clinically upon examination,
without excluding them you will KILL a person by homicide, or
criminal negligence, despite the reversibility of brain damage.

Brain death is defined as the irreversible cessation of
function of the entire brain with three specific criteria: 1) coma,
2) absent brainstem reflexes and 3) apnea. In addition to these
clinical criteria, there are important prerequisites: 1) NO
intoxication or poisoning, 2) NO core temperature greater than 32
degrees Celsius,3) clinical or neuro-imaging evidence of acute
central nervous system catastrophe and 4) absence of confounding
medical conditions such as severe electrolyte, acid-base, or
endocrine disturbances.

As a safeguard in determining
brain death a number of tests need to be carried out every 6 hours
and recorded, the physicians performing this determination must not
be part of a transplantation team. In some cases, 48 to 72 hours is
required to evaluate brain death and a repeat examination with
observation up to an additional 24 hours is sometime needed. The
length of time between serial examinations to declare brain death
varies marginally from 6 to 72 hours.

Notably the Sudbury
doctors involved were a part of an organ harvesting and
transplantation team. It was Dr. Sauve who, utilizing deception,
sought to obtain permission from a remote party to obtain the
victim's eyes, without immediate family knowledge or consent.

With
respect to the initial CT scanhereinbefore mentioned, according to
the coroner's expert "in the right occipital region there is a
spot that measures less than 1 cm that is consistent in appearance
with either a small hemorrhage or perhaps a small metastatic tumor".
He could only speculate. NO biopsy was done (a biopsy, is required
for obtaining tissue for pathological confirmation of the diagnosis).
The solitary lesion is also consistent in appearance with an abscess
secondary to an occipital dermoid cyst, or early stage cerebritis
during/after capsule formation in the early stage of abscess
development. Rupture of a dermoid and leakage of a cyst contents into
a ventricle or subarachnoid space may produce an epidymitis or
meningitis respectively. Further, capsules can rupture resulting in
the formation of multiple abscesses.

The bald truth is
that localizing signs of brain tumor include a loss of visionon the
side of an occipital neoplasm. Compare occipital abscess, or pyogenic
brain abscess, usually of bacterial origin. The occipital lobes
interpret vision. Brain tumors are more solid/dense and therefore are
usually associated with multi-focal deficits; tumors of the occipital
lobe usually produce homonymous hemianopia or partial visual field
deficits. Had the lesion been a recent tumor, there would have been
onset visual misperception in half of one or both visual fields, with
visual impairment and subsequent loss of vision with evolution, prior
to hospital admission. That did not happen.

Know that even
multiple brain abscesses may not cause focal deficit to suggest their
presence. Non-neoplastic demyelinating processes of the brain with
ring enhancing lesions and mass effect on MRI imaging, mimicking
malignant brain tumors, are rare phenomena. The radiologic appearance
of demyelinating pseudotumors as contrast-enhancing solitary masses
that mimic tumor is well documented, ie., "tumor-like"
masses of demyelination, or granulocytes that mimic brain tumors
(granulocytes, which are cancer killing cells turn black as they
die), or infection. Further, with multiple abscesses or infection the
meninges typically show a purulent exudate that obscures the sulci
making radiographic appearance of microabscesses less visible, hence
they are "not well opacified". Morbidity due to a brain
abscess generally results from brain herniation due to mass effect,
the result of iatrogenic neglect, or substandard care.

No
autopsy was performed. Further, a family request for a formal inquest
was also denied. Dr. McLellan concluded that Arlene Berry had died of
natural causes suggestive of metastatic CA of the brain with
multiple brain tumors after eliciting what can only be construed as a
most questionable opinion from one of his fellow colleagues believed
to be associated with the Sunnybrook Health Sciences Centre, where
McLellan spearheaded the North Telehealth Network. However, the
medical record of Arlene Berry for May 23rd and 24th of 2000, tells a
very different story from the opinion postulated.

Eleven
percent of mass lesions in cancer patients are not metastases; mass
lesions that can masquerade as brain metastasis include abscess(20%)
and granuloma (less common and mostly associated with mycobacterial
or fungal infection). The commonly observed deficits observed in CNS
infection include weakness on one side of the body (hemiparesis),
impaired speech production (dysphasia), visual field deficits (may or
may not be present), and an inability to smoothly coordinate muscle
movements, such as during walking (ataxia).

Patients with a
dioagnosis of primary or metastic brain tumor(s) associated with a
CNS event should have a meticulous review of their history for other
possible causes. Brain Tumor is often considered in the diagnosis of
lesions demonstrated on brain computed tomography (CT) and/or
magnetic resonance imaging (MRI). Brain tumors usually show abnormal
densities on CT or altered signal intensities on MRI, mass effect,
and sometimes contrast enhancement after the intravenous
administration of contrast material. Lesions with these features,
however, are not always brain tumors and establishing the diagnosis
of a brain tumour is not always a straightforward process.
Cerebrovascular diseases, demyelinating processes, inflammatory or
infectious diseases, and other miscellaneous diseases can show
similar imaging findings.

Many non-neoplastic neurological
diseases can mimic brain neoplasms on neuroimaging or on histological
examination, including multiple sclerosis, stroke, pyogenic abscess,
toxoplasmosis, tuberculosis, cysticercosis, fungal infections,
syphilis, sarcoidosis, Behçet disease, radiation necrosis,
venous thrombosis, Guillain Barre syndrome and others. Multiple
intracerebral hematomas can also mimic brain metastases. West Nile
virus encephalitis mimicking central nervous system metastases from
small cell lung cancer is reported in the literature.

The
investigation and analysis has been ongoing for almost 9 years with
everything pointing to possible GBS, with two second-guess
possibilities in the differential which can not be ruled out, namely
West Nile Virus, or Botulism, in that order. The differential
diagnosis can be difficult, because the symptoms mimic those of
Guillain-Barre syndrome. Further, fever in WNV apparently does not
present in all cases. With GBS, fever is usually absent at onset.
Although brain death mimickery is not an acute finding in WNV, it is
with the Miller Fisher variant of GBS, a brainstem encephalitis which
also mimics brain death. That botulism may also mimic brain death
needs always to be kept in mind. Yet, meningitis, encephalitis and
Guillain-Barre syndrome remains undeniably the presentation in this
case. I have to ask, is there a common denominator here?

Meningitis and encephalitis are usually caused by
viruses or bacteria. Less commonly, encephalitis can result from a
bacterial infection, such as bacterial meningitis, or it may be a
complication of other infectious diseases. Guillain-Barré is
preceded by a viral or bacterial infection. Viral meningitis is the
most common cause of aseptic meningitis, an inflammatory process
involving the meninges in which usual bacterial etiologies cannot be
identified.

Questionable
Opinion:

The opinionated author
(whose signature was erased) perceives himself to be expert in his
field. Believed to be a neurosurgeon, he is generally lacking. The
bald truth is that specialists in a given field are not always expert
in that field. For the record, his opinion remains unsubstantiated.
He also failed to provide any evidence whatsoever which might support
a finding of metastatic CA of the brain. Further, a paltry CT scan
does NOT provide conclusive proof of metastatic brain tumors. For
that it takes a biopsy and in this case that was never done.
Therefore the true nature of the lesions were never established. Nor
does a CT provide conclusive proof of brain death. In fact the author
who rendered the opinion suggesting metastatic cancer is pretty much
off-the-wall; what one might expect from a "hand-picked"
self-interest cash for comment shill, or someone to one equated with
a first year medical student who is thus unworthy of belief.

Further, the opinions expressed by the nameless
author admits that his opinion "does not
take into account all the facts and circumstances surrounding the
patient's death", being his disclaimer.
Such a disclaimer also raises doubt about the accuracy of the
opinion. He bases his opinion on assumption, while ignoring the
facts. Obviously he did not take the time to study the patient's
medical record. Instead he opted to tailor or lard his report to
justify the opinion that was, in his opinion , being sought by Dr.
McLellan for the sake of expediency. This infers a blatant attempt by
all concerned to obfuscate the truth. A finding of possible
meningitis with abscess of the brain and/or multiple intracerebral
hematomas would have given more credibility to his opinion. In fact,
some of the opinion expressed by the nameless author could also be
used to support a finding of meningitis. There is absolutely
nothing on record to support a finding of metastatic cancer of the
brain, apart from a paltry CT, with no evidence on record to support
it.

Rapid deterioration is an invariable accompaniment of an
untreated condition, in this case, an undiagnosed case of fulminant
GBS with overlaping meningitis, in which rapid progress of the
disease may actually be displaying a pronounced"blood-brain
barrier breach", characterized clinically by "rapid
evolution", the result of totally inappropriate treatment, and
medications which can breach blood-brain barrier integrity, ie. the
drug Stemetil. Breakdown of the blood-brain barrier usually precedes
inflammatory demyelination. GBS is often associated with
cerebrospinal meningitis and encephalitis. Coma in GBS is rare and is
the symptom of cerebrospinal meningitis and encephalitis. The
clinical manifestations of this condition included areflexia in the
cranial and spinal nerves as well as apnoea. Cisterns may contain pus
in cases of meningitis or other inflammatory conditions, such as
sarcoid, or demyelination.

Any medical professional who, in
rendering his opinion as to a cause of death, volunteers
his unsolicited opinion as to another
doctor’s conduct or standard of care
in the face of overwhelming evidence to the contrary, finding in
favour of his fellow colleague(s), invites
being labeled retrospectively with the most
nefarious motives.

Although two physicians with experience and expertise
must be responsible for the declaration of brain death, and a
neurologic condition capable of causing brain death is a mandatory
prerequisite to the diagnosis of brain death, there are reports in
the literature of conditions that mimic brain death or that
provide examples of the mistaken diagnosis of brain death.

The clinical diagnosis of brain death
occurred after the patient had received an IV tranquilizing agent and
while still under the influence of morphine sulfate, an opiate
narcotic.

GBS is one
of the few neurological diseases whose clinical manifestations may
mimic or appear to be identical to those in brainstem death,
illustrating an extreme polyneuropathy.

Canadian
standards do not test function of the "entire brain", and
there is sound evidence that many individuals who meet the clinical
criteria of brain death continue to have some cortical, subcortical,
or brain stem function. Many patients diagnosed as brain dead do not
have hemodynamic collapse, they have physical findings such as bowel
sounds, and are reported to have autonomic reflexes (tachycardia and
hypertension) at the time of organ retrieval, suggesting a horrific
death.

The clinical diagnosis of brain death in this
case was made in the presence of metabolic derangements and endocrine
abnormalities and constitutes an act of wanton and reckless disregard
for human life. There have been many challenges to the several
concepts of "brain death" and the means of their diagnosis
worldwide (vide infra). Indeed, it seems that there is now an
emerging consensus that "brain death" diagnosed by any of
the protocols in current use worldwide is "not death."

After
the diagnosis of brain death, the focus of patient care shifts from
interventions aimed at saving the patient's life to interventions
aimed at maintaining viability of potentially transplantable organs.
Given that current clinical testing does not assess subcortical brain
function, ‘whole brain death’ cannot be conclusively identified
at the bedside by using clinical criteria, and most certainly not on
the basis of a paltry CT scan. The CT is useful only in pretty severe
cases, such as head trauma and/or during the few days after an anoxic
(lack of oxygen) brain injury and also in cases where there has been
a massive stroke. If the question is ischemic injury [brain damage
caused by lack of blood/oxygen to part of the brain] you want an MRI
and PET. For subsequent evaluation of brain injury, especially brain
death, the CT is pretty much useless.

Many Ontario physicians
actively involved in the identification of brain death, if the truth
be known, are unable to identify the requisite diagnostic components
of brain death, and/or are unable to apply the criteria correctly.
Some physicians will take advantage of the situation to perpetrate a
medical murder in order to harvest organs from potential donors,
while others will simply declare "brain death" in order to
avoid liability for medical wrongdoing.

Organ transplantation
is premised on professional and public acceptance that the donor is
dead and that the cessation of all brain function persists for an
appropriate period of observation. The diagnosis of brain death
allows organ donation or withdrawal of life support. The diagnosis of
brain death is dependent upon the exclusion of certain medical
conditions without which the diagnosis of brain-death cannot be
considered and influence of medications is one of them.

Brain
death declaration may be described as an esoteric creation of
neurologists and neurosurgeons who are seeking to speed up death for
the purposes of an organ transplants. In this case, the harried and
hurried physicians did not rely on those esoteric criteria in
pronouncing the patient "dead." They simply used the
questionable "brain death criteria" (based on a paltry CT
after withdrawal of ventilator support) as a pretext to operate and
remove their patient's eyes while she was still very much
alive.

There are numerous reports of brain death declaration
and at least two where the criteria were deliberately misrepresented,
or obfuscated in an attempt to retrieve an organ for transplantation,
as in this case.

Brain
death:
resolving inconsistencies in the ethical declaration of ...
Purpose:
The first criteria for
the determination of brain
death were
developed ... two
cases were
presented where
the criteria
were deliberately
misrepresented in
...
www.cja-jca.org/cgi/content/full/50/7/725

More
pages from cja-jca.org

Status
Code 0, or No Code.
In my opinion, Arlene Berry's death was premeditated, as evidenced by
the physician's documented “Patient Status
0" (code 0), and Dr. McLellan
knowingly returned a false finding for which he remains to be held to
account. From the facts of this case it seems clear that this moral
compass isn't such a straight arrow after all.

In a letter to
the College of Physicians and Surgeons of Ontario dated November 28,
2000, Dr. Jordan writes "discussed the
situation with family members and a decision was made to intubate Ms.
Berry", while the Ambulance Call Report
seen at N-7 documents an unsigned patient "Status
Code 0", or endorsement of a No Code by
proxy. This is an order akin to a 'DNR' denying
medical intervention, in this case by
passing-the-buck together with all the possible blame.

Patient
condition "Status Code Zero"
taken from CMAJ articles, is used to describe a "dead"
patient condition. The Yale Law Journal, Vol. 93, No. 2 (Dec., 1983),
pp. 362-383, criticizes questionable hospital policies "that
resort to third party adjudications in No-Code decision making",
and provides insight into the assigning of a "no-code
order" instructing personnel "not
to attempt resuscitation", as in this case. Looking over the
chart it is clear that obtaining a 'no code' status in the face of
immune mediated adversities by reason of Dr. Jordan's and Dr.
Spiller's failure to recognise and treat an emergency situation
accordingly, and by reason of Dr. Jordan's
failure to attend in a timely manner was the
next essential step for these two negligent physicians in executing
Arlene Berry's death in order to avoid liability issues.

The Criminal Code of Canada regards euthanasia,
whether passive or active, as culpable homicide, or murder: A
culpable homicide is defined as murder "where the person who
causes the death of a human being means to cause his death" (s.
229, Criminal Code of Canada). Unless the law has changed in recent
years, euthanasia carries a fixed, minimum penalty of 10 years in
prison. The 'no code' as evidenced in this case by a "Status
Code 0" was ordered by Drs. Jordan, and Spiller, without family
knowledge or consent when the patient's condition began to rapidly
deteriorate following a whole chain of medical negligence and
substandard care that is nothing short of criminal.

With the
help of Dr. Mark Arthur Spiller (who admitted Arlene Berry to the
Kirkland and District Hospital on the evening of May 23rd), a
cross-covering physician working the ER with Dr. Jordan on a
rotational basis, these two unscrupulous physicians were able to
recruit two of their equally unscrupulous Sudbury colleagues to
complete their dirty work at arms length. Interestingly, Dr. Spiller
had been a local appointed coroner working under Dr. McLellan at the
time, and also a classmate of Dr. Stephane Sauve, one of the Sudbury
doctors involved in this medical homicide.

Although it is
clear that Arlene Berry was transferred to Sudbury with ventilator
support, and although Drs. Jordan and Spiller were aware of the need
for emergency care and life support, after ordering it, they canceled
it, using the secretive no code (Code 0) endorsement as a pretext for
evoking a declaration of death and in fact waited for the patient's
death.

Within a few hours following her transfer from the
Kirkland and District Hospital to the Sudbury Regional Hospital
Arlene Berry was declared as having met with 'brain death criteria' ,
while under the care of Drs. Sauve and Adegbite. Her remains were
kept in Sudbury for several days prior to being returned to Kirkland
Lake. Withholding life sustaining treatment from an "undiagnosed"
patient with concurrent hyperglycemia, hypokalemia and electrolyte
abnormalities in combination with a severely paralyzed motor function
and who is under the influence of sedative hypnotic and tranquilizing
agents is of questionable legality. Death results from respiratory
paralysis and subsequent asphyxiation. Brain death is what happens
when ventilator support is discontinued.

Turning off a
respirator is a form of passive euthanasia that is practiced by
doctors with a family's consent. Turn off the respirator and in the
natural course of affairs the patient dies from lack of oxygen. To
practice euthanasia by withdrawing life support to a critically ill
patient is a medical homicide (to kill or destroy by preventing
access of air or oxygen). Passive euthanasia involves an allowing of
"nature to take its course". Active euthanasia consists of
killing someone (to do acts causing death), or by choosing not
to act is also an act, which determines the course and the outcome of
events.

An act of wanton and reckless disregard for human
life is an act of criminal negligence, in this case, causing death.

In looking over the coroner;s report, was this Dr.
McLellan's way of exonerating those responsible for Arlene Berry's
death in order to shield the local appointed coroner (working under
him at that time) from liability? Or was it just a blatant attempt on
his part to shield his fellow colleagues from absolute disgrace?

1. Dr. Sauve sought to open the way to organ donation
from brain death under misleading conditions (influence of
medications, and metabolic disturbances), further utilizing deception
to obtain remote third party consent to harvest my wife's eyes,
bypassing permission from the immediate family, namely her defacto
spouse and children.

2. Interestingly, Dr. Sauve was a
"classmate" of Dr. Mark Spiller, having attended the
University of Toronto, Class of '89. Dr. Sauve's name appears on the
Clinical Investigator Inspection List (CLIIL) for Investigational New
Drug Studies, and also on the Population Health Research Institute
Canada Center Information list, suggestive of perhaps some hidden
agenda, either in the nature of influencing government spending for
hospital funding, or some sinister plot involving population control
by medical homicide, or both.

3. Suspected of theft of organs
for harvesting from a living person by misrepresentation and
accessory to medical homicide are: Adegbite, Andrew Babafunso
Olanrewaju - Sudbury, ON - (CPSO#54992 ) and Dr. Stephane Jean Sauve
- Sudbury (CPSO# 61381); MDs with privileges at the Sudbury Regional
Hospital - Laurentian Site.

4. Suspected of criminal
negligence causing bodily harm, and liability murder in order
to save face are Jordan, Edward Henry - Kirkland Lake, ON
(CPSO#61732), and Spiller, Mark Arthur (CPSO#60977); cross-covering
physicians working the ER at the Kirkland and District Hospital in
Kirkland Lake, ON. In concert with the attending physician, the
decision was made by Dr. Jordan to have the patient's senses blunted
and prepared for organ harvest.

4. Suspected of conspiracy to
conceal an indictable offence(s) and returning a false finding is Dr.
Barry Alexander McLellan who so happened to be the Regional
Supervising Coroner at the time. Whatsmore, he had been Dr. Spiller's
immediate supervisor. A family request for a formal inquest was
boldly denied by Dr. McLellan. Interestingly, Dr. McLellan's office
was in charge of administering the Human Tissue Gift Act and the
Anatomy Act transplants. Dr. McLellan went on to become Ontario's
chief coroner and discredited pathologist Charles Smith in order to
turn suspicion away from his own faulty findings and sordid
immorality. As of September 17, 2007, Dr. McLellan joined Sunnybrook
Hospital in Toronto as its new CEO. It is not yet clear whether
McLellan stepped down in order to seek refuge among his fellow
equally contemptible colleagues at Sunnybrook, or if he was simply
given the boot.

This case is now a matter of public record.
It has been widely reported and publicized through various Internet
media, Topix forums as well as the Indymedia IMC throughout Canada,
the USA, and the UK. The long standing silence of the doctors
named herein confirms their active and ongoing concealment, which
includes not only secrecy and deception, but also reticence and
non-acknowledgment for their part in the Arlene Berry death cover-up.

The doctors must have intended the death to occur. It is
never permissible to take any direct action designed to kill a
patient. If a doctor deliberately let a patient die who was suffering
from a curable illness, the doctor would certainly be to blame for
what he had done, just as he would be to blame if he had needlessly
killed the patient. Charges against him would then be appropriate. If
so, it would be no defense at all for him to insist that he didn't
"do anything." He would have done something very serious
indeed, for he let his patient die.

To allow or hasten a
patient's death to cover-up error or negligence, or to obtain organs
is reprehensible. It is nothing short of murder since such an act or
omission which causes death carries the "intended"
consequence of the act or omission, hence, the mens rea or criminal
intent.

In this case the intent, evidenced by the
"Status Code 0" seen at N-7 of the record, is clearly an
act of intending to cause the patient's death.

View the medical record of Arlene Berry

DaNgeR--DNR!!!

Brain
death:
resolving inconsistencies in the ethical declaration of death

GUILLAIN
BARRE
SYNDROME (GBS)

Guillain
Barre
syndrome | NowPublic News Coverage

Guillain-Barré
Overlap
syndrome: Signs & Symptoms

Guillain-Barré
Syndrome:
The Untold
Story | NowPublic News

Encephalitis:
Diagnosis and Treatment

Hidden
Agenda: Behind the Scenes

RateMDs.com
- Dr. Edward
Henry Jordan
Doctor Ratings

Re:
Andrew Adegbite,
MD.# 54992 Criminal Negligence

Sudbury
Regional Hospital - Andrew Adegbite,
MD - Topix

Dr.
A.
B. Adegbite
- Sudbury, ON Doctor Ratings | RateMDs.com

Bacterial
Gastrointestinal
tract (GIT) infections

Infections
by Organ System

Medical
records for attorneys

Medical Dictionary Online-Medical-Dictionary.org

Arlene Berry: Patient Record

On Bad Doctors...

2009-05-06T15:39:00.000-07:00

Are Dr.s evil, stupid or what? Bad Doctors

Questionable Doctors...

2009-05-06T14:31:00.000-07:00

The physician who cannot tell that his drugs are producing ill effects upon his patient until serious, even fatal consequences evolve, has no business prescribing drugs, or practicing medicine for that matter.

You either have it or you don't.

Stories of the Incredibly Stupid

Truth & Justice Demanded

2009-04-30T16:40:00.000-07:00

A PHYSICIAN CANNOT ESCAPE the essential principle: Primum non nocere "First Do No Harm". Whenever you grant immunity from fault you breed irresponsibility.

Doctors Forcing Patients to Sign Gag Orders: Undue influence

2009-04-28T18:27:00.000-07:00

"Can you believe this?!

There are doctors who are forcing patients to sign a contract promising not to criticize the doctor, "his expertise and/or treatment."

No signature-No medical care

If the patients won’t sign the contract, the doctors won’t treat them.

I will ask again: Can you flippin’ believe this?!

Doctors don't like online reviews

The contracts are in response to websites that are springing up around the internet that allow patients to post reviews about their doctors. Doctors don’t like the fact that the websites allow patients to post negative comments, but doctors have no way to respond without breaching patient confidentiality.

Blackmail?

So they have resorted to blackmail. Sign the paper or don’t get medical care!

Can you believe this?!"

SOURCE: RateMD's

Undue influence

Know that virtually any act of persuasion that over-comes the free will and judgment of another, including exhortations, importunings, insinuations, flattery, trickery, and deception, may amount to undue influence. Undue influence differs from duress, which consists of the intentional use of force, or threat of force, to coerce another into a grossly unfair transaction. Blackmail, Extortion, bad faith threats of criminal prosecution, and oppressive Abuse of Process are classic examples of duress.

Four elements must be shown to establish undue influence. First, it must be demonstrated that the victim was susceptible to overreaching. Such conditions as mental, psychological, or physical disability or dependency may be used to show susceptibility. Second, there must be an opportunity for exercising undue influence. Typically, this opportunity arises through a confidential relationship. Courts have found opportunity for undue influence in confidential relationships between Husband and Wife, fiancé and fiancée, Parent and Child, trustee and beneficiary, administrator and legatee, Guardian and Ward, attorney and client, doctor and patient, and pastor and parishioner. Third, there must be evidence that the defendant was inclined to exercise undue influence over the victim.

This section is from the book "Handbook Of The Law Of Contracts", by Wm. L. Clark, Jr.. Also available from Amazon: Handbook of the law of contracts.

Influence

145. Undue influence is a species of fraud. It may be said generally to consist -

(a) In the use by one in whom confidence is reposed by another, or who holds a real or apparent authority over him, of such confidence or authority for the purpose of obtaining an unfair advantage over him.

(b) In taking an unfair advantage of another's weakness of mind.

(c) In taking a grossly oppressive and unfair advantage of another's necessities and distress.

146. EFFECT. Undue influence renders a contract voidable at the option of the injured party.

The Questionable Cases Of Dr. Charles Smith

2009-04-25T15:02:00.001-07:00

Pathologist admits to being self-taught - A Review

Dr. Charles Smith was once considered top-notch in his field of forensic child pathology. In 1999, a Fifth Estate documentary singled him out as one of four Canadians with this rare expertise. A review of discredited pathologist Charles Smith has found he made errors in 20 child autopsies, 13 of them resulting in criminal charges.

An inquiry led by Justice Stephen Goudge and concluding in October 2008, found that Smith "actively misled" his superiors, "made false and misleading statements" and exaggerated his expertise. Dr. Smith had provided an opinion regarding a number of cases where cause of death that was "not reasonably supported by the materials available for review". That would not be unusual, doctors do it all the time and Ontario's chief coroner is no exception.

TheStar.com | Ontario

"The review is a welcome reminder that we should never put experts at the same level as God."

The Questionable Cases Of Dr. Charles Smith

2009-04-25T15:02:00.000-07:00

The Questionable Cases Of Dr. Charles Smith: A Review

TheStar.com | Ontario

"The review is a welcome reminder that we should never put experts at the same level as God."

From Cancer QUACK To Chief Coroner

2009-04-25T14:11:00.000-07:00

Barry A. McLellan MD (Medical Dolt) LIAR

Dr. Barry McLellan was the Chief Coroner for Ontario. Prior to this position, he was the Deputy Chief Coroner for Forensic Services, having left his position as director of the NORTH Network, an experiment undertaken by the Harris government from a 1995 OMA study to compensate for hospital funding cutbacks, and doctor shortages.

The NorthNetwork has a vested interest in protecting itself against the legal pitfalls of partnership doctors treating their patients at arm's length, over the telephone, unseen, which is exactly what happened to Arlene Berry, a 41 year old mom with two young children who was treated over the telephone unseen and died less than 24 hours later. The event took place in the year 2000. A private investigation first yielded possible evidence of multiple brain abscesses, or demyelination likely masquerading as brain metastasis with evidence of possible iatrogenic drug induced meningitis based on the deceased's blood values. Certain portions of her medical record had been altered and falsified in order to cover-up a whole chain of medical negligence, which hampered meaningful investigation. No autopsy was performed. A family request for a formal inquest was also denied by Dr. McLellan. It took almost eight years of private investigating to finally uncover the truth, matching virtually all of the residual evidence to overlapping variants of the Guillain Barre syndrome. Possible wound botulism is the only logical differential making any sense at all, since Miller Fisher variant of GBS closely mimics botulism. One thing is certain and that is that Arlene Berry did not die as suggested by Dr. McLellan.

Arlene Berry was transferred from Kirkland Lake to Sudbury with an unauthorized "Status Code 0", a No Code order akin to a DNR, or withdawal of life support. According to Sudbury doctors, Arlene Berry died meeting brain death criteria. Guillain Barre syndrome is known to mimic brain death. This is a clear cut case of doctors "acting with wanton and reckless disregard for human life" and is the hallmark of criminal negligence causing death.

Dr. McLellan had concluded that Arlene Berry had died of natural causes suggestive of metastatic CA of the brain with multiple brain tumors, after eliciting an opinion of one of his fellow colleagues from the Sunnybrook Health Sciences Centre where he had lectured. The medical record of Arlene Berry for May 23rd and 24th of 2000 tells a very different story. In my opinion, Dr. McLellan knowingly returned a false finding.

Case in Point: SERIOUS breach of standard which goes to impeach the credibility of the Coroner's investigation into the unnecessary death of Arlene Berry. Further, Dr McLellan was Vice-President of Medical Trauma and Clinical Services at Sunnybrook Health Science Centre in Toronto Ontario for 5 years. He was medical director of the NORTH Network and had recently assumed the position of Regional Coroner. A question has arisen with respect to his "relationship" to Dr. Mark Spiller?

A family request for a formal inquest into Arlene Berry's death elicited to following response from Dr.McLellan: "I want to stress that an inquest is not intended to be the vehicle by which someone is found to be responsible or accountable ...". As a result of my investigation and having carefully reviewed all information available I do not feel.. that a jury might make useful recommendations directed to the avoidance of similar circumstances". . "The venue to determine accountablity is either the criminal or civil courts". . "After careful consideration of all information available to me I have therefore made a decision to not hold an inquest into Ms. Berry's death ".

Further, Dr. McLellan had told the deceased's family that he had no dealings through his office with the College of Physicians & Surgeons. He "lied", in fact he conducted what Dr. Jordan's legal counsel described as a "parallel investigation" with "multiple communication" between the Coroners' office and the College. The Coroner had seized the medical records for almost a year before family was allowed to get them. The KDH would not release them to us without Dr. McLellan's approval even after Dr. McLellan had completed his investigation. The Coroner possesses superior knowledge or the means of discovering discrepencies in the medical record which he either ignored or deliberately withheld from the police - turning a blind eye that which should have been obvious or suspicious. The feeble-minded doctor thought he could pull a fast one, never suspecting that I would go as far as I did to prove him a liar.

Dr. Barry McLellan was one of the original proponents of the NORTH project, leaving his position as medical director of the North network to become Regional Coroner for Northeastern Ontario. In fact, he was affiliated and closely tied to all of the doctors and hospitals named in the "Arlene Berry Death Coverup", all of whom were partners in the North telehealth network. As such, Dr. McLellan had a personal and vested interest in the Arlene Berry case as to affect his personal judgment. He allowed his professional duty to come into conflict with his personal interests as to constitute a conflict of interest, ahead of public duty, which he misused for personal ends. Conflict of interest is a precondition for biased or corrupt behavior. He had a duty to disclose such interest(s) and by failing to do so he acted illegally, and in my opinion, contrary to the provisions of the Criminal Code of Canada, via provisions related to corruption in public office, and the Conflict of Interest Code, breach of trust and public endangerment by turning a blind-eye on medical wrongdoing in order to protect a local appointed coroner, Dr. Mark Spiller, working under him at the time. Dr. Spiller also so happened to be the admitting physician in this case was just as negligent as the deceased's family physician, Edward Jordan in treating their patient as routine in the face of life threatening indicators and finally ordering her death to cover up their collective medical stupidity.

Dr. McLellan graduated from the University of Toronto in 1981, a fraternity of "liars" which has abdicated its responsibility for teaching ethics and proper patient care to concentrate on "teaching medical students to lie" (something Dr. McLellan is very good at), where students are actually graded on how well they tell the lies, and where the long term benefits accrue to keeping each other out of jail. But the system has a way of promoting incompetence. McLellan achieved the position of chief coroner for Ontario based on the Peter Principle concept.

The bald truth is that the Coroners Office has for years been used as a dumping ground for cases embarrassing either to the government or the medical profession. Interestingly, Dr. McLellan went on to become Ontario's chief coroner and discredited pathologist Charles Smith in order to turn suspicion away from his own faulty findings. Much the same as the pot calling the kettle black, a classic example of the "peter principle" at work, both doctors promoted far beyond their competency levels. As a result, they are scared stiff over the prospect of being discovered and sooner or later one them does it to the other to disguise his own inadequacies.

"The theory that employees within an organization will advance to their highest level of competence and then be promoted to and remain at a level at which they are incompetent." ... "In a hierarchically structured administration, people tend to be promoted up to their level of incompetence, like Dr. Charles Smith. And like Smith, Dr. McLellan also provided an opinion regarding a case where cause of death was "not reasonably supported by the materials available for review".

This page is under construction. Please check back as more information is added.

"Nobody made a greater mistake
than he who did nothing because he
could do only a little." (Edmund Burke,
political thinker

What Ever Happened to First Do No Harm?

2009-04-25T12:31:00.000-07:00

"The healthcare industry has done a dreadful and shameful job of policing itself. The medical boards are slow to ever discipline doctors and the "peter principle" is alive and well in the healthcare industry.

Physicians have adopted the corporate health care mentality; where a patient is more a statistical profit margin than a "sick patient". Patients are treated to ten minutes and a cloud of dust from their physicians; rather than a caring, interested, healer." - John Hopkins

A doctor is history's worst serial killer

2009-04-24T20:21:00.000-07:00

The difference between God and a doctor is "God doesn't think he's a doctor."

Harold Shipman , Doctor Death

Dr. Harold Shipman

How system helps shield bad doctors

2009-04-24T18:10:00.000-07:00

May. 5, 2001. 07:48 AM

How system helps shield
bad doctors
College admits flaws in process

ROBERT CRIBB, RITA DALY AND LAURIE MONSEBRAATEN
STAFF REPORTERS

Medical Secrets

You've just been diagnosed with cancer. You're scheduled for surgery in a few weeks by a doctor you hardly know.

In Ontario - and across Canada - provincial laws governing doctors say you can't find out if your surgeon has been hauled before the physicians' watchdog for complaints of malpractice, even for the exact operation you're facing. You aren't told if the surgeon is under investigation now, or has a spotless record.

The medical complaints system is cloaked in secrecy.

Since discipline procedures at the College of Physicians and Surgeons of Ontario can take three years, some dangerous doctors continue practising without patients knowing there may be a risk.

In the last six years, the college investigated 13,000 complaints about doctors. Of those, 99 per cent were either dismissed outright or handled internally and in secret.

We can learn nothing about them. The college, which says it is bound by privacy provisions in the Regulated Health Professions Act, will not reveal the names of the doctors involved, how many had complaints against them, the nature of the complaints or how they were handled.

The only window into a doctor's competence, integrity and past professional conduct is a public disciplinary hearing conducted by the college.

A Toronto Star investigation shows that since 1994, when new legislation governing doctors came into effect, only 141 complaints have reached this stage.

Of 13,000 complaints lodged against doctors, 99 per cent were either dismissed or handled in secret by the College of Physicians and Surgeons of Ontario

Data on those cases, compiled by The Star using the college's public records, reveal a disciplinary system that typically hands out lenient penalties, rarely revokes licences, can take years to render decisions and puts doctors on higher legal footing than complainants.

The Star's data show that 111 doctors have been found guilty of offences ranging from fraud, drugging and sexually assaulting patients and missed diagnoses, some causing death. Of those, 34 received the college's maximum penalty - licence revocation.

The majority of sanctions handed out were suspensions averaging less than three months or reprimands, usually with some conditions, such as cash fines, additional training or letters of apology.

Ontario's college took an average of three years to reach those decisions - tied with Manitoba as the slowest medical disciplinary system in Canada.

The overwhelming majority of Ontario's 20,480 practising doctors - skilled professionals dedicated to meeting the highest standards of care - has never been the subject of a college disciplinary hearing. They may face a minor complaint or two over the years but for the most part their records are clean.

But some physicians who have done great harm are allowed to continue practising.

Dr. Kenneth Bradley, a former Milton chief surgeon, was twice was found guilty of medical negligence involving patients who died. Still, Bradley kept his licence and later moved to Virginia, where he was implicated in another four deaths.

Dr. Stanley Dobrowolski, a London, Ont., psychiatrist, was disciplined three times for sexually related offences. He is still treating patients.

Dr. Anthony Laws, who practises in Oakville, falsified documents to cover up his role in the death of a 14-year-old patient under his care.

People who feel they have been harmed by a physician can seek damages through the courts. But the self-governing College of Physicians and Surgeons of Ontario is the only authority that can suspend or revoke a doctor's licence.

The Ontario Medical Association, which speaks for the province's doctors, believes the college is doing an ``exemplary job'' of protecting the public and guiding the profession.

Dr. Alex Schumacher, the OMA's immediate past president, says the low discipline rate is a reflection of the province's high licensing standards compared to other jurisdictions.

But the college itself acknowledges the system should be more open and must resolve patient complaints more quickly.

`We have to act before the public loses confidence.'

- Dr. Rocco Gerace
President of the College of
Physicians and Surgeons of Ontario

``We have to act before the public loses confidence,'' says college president Dr. Rocco Gerace.

And that means getting tougher on dangerous doctors, say some college members.

``Clearly there has been less than effective identification of those (doctors) who have serious problems that need addressing,'' says Dr. David Walker, dean of health sciences at Queen's University and co-chair of the college's complaints committee.

Last year in Ontario, 21 doctors had their licences revoked or suspended.

During the same period, Michigan, with a similar number of physicians, suspended or revoked the licences of 102 doctors. In Massachusetts, another comparable jurisdiction, it was 45.

In Ontario and elsewhere, most doctors can keep practising while they are being investigated. But in a growing number of states, information about doctors under investigation is posted on Web sites for anyone to see.

Gerace doesn't think the American focus on discipline gets to the heart of the matter.

``Their measure of success is their percentage of discipline referrals. I would suggest to you that a discipline referral is a failure.

``We have to balance the will of the public for revenge - an eye for an eye and a tooth for a tooth - versus what's best for the public interest, which is having doctors deliver better care. Do we need to have a few examples out there? Just beat someone up?''

Gerace says that while U.S. jurisdictions rely solely on discipline, Ontario has developed unique programs to assess and retrain doctors whose skills are lacking.

The philosophy reflects the college's belief that most doctors who have made clinical mistakes are anxious to improve and will become better doctors through education rather than punishment.

But according to a recent government report about the discipline process, there is no system in place to ensure this approach is actually working.

The government-commissioned report expressed concerns that the quality assurance program ``may not be the appropriate response'' for patient complaints about doctors.

And since the process is handled entirely behind closed doors, there's no way patients or the public can judge whether their complaint was handled adequately.

That secrecy is only lifted when complaints trigger disciplinary hearings.

In the past six years, the majority of those hearings - 77 out of 141 - concerned sexual misconduct, followed by cases involving patient deaths (19), and psychological harm inflicted by doctors (10).

Other offences that went to disciplinary hearings included fraud, physical injuries and faulty record keeping.

The focus on sex-related cases reflects the college's zero-tolerance policy, adopted in 1994 following a provincial government task force that estimated 10 per cent of Ontario doctors had sexually abused a patient at some time.

The Star data show that 25 of the 61 doctors found guilty of sex-related complaints had their licences revoked, 24 received suspensions averaging 3* months, 10 received reprimands and two quit their practices.

Zero tolerance covers anything from inappropriate touching to consensual intercourse to rape. The Ontario Medical Association is currently challenging this policy before the courts as a Charter of Rights and Freedoms issue.

The Star data shows that in at least four cases, physicians allowed to continue practising went on to repeat the same offence.

Dobrowolski, the psychiatrist from London, Ont., is a prime example.

He has appeared before the college three times since 1994 on complaints ranging from hugging, kissing, fondling and sexual intercourse with patients.

At his second appearance, in 1996, the panel concluded Dobrowolski demonstrated ``an extremely worrisome pattern of behaviour.''

Dobrowolski received a short suspension and two reprimands but never lost his licence. He continues to treat patients in London.

``It (the disciplinary system) is such an unpleasant process from the point of view of a physician that even just talking about it brings back bad memories,'' he said, when contacted by The Star. ``You try and put it behind you and get on with life.''

Many doctors see the college as an adversary that often dishes out unwarranted punishment.

Of the 19 cases of death under physician care that reached the disciplinary committee since 1994, no fault was found in five.

In the other 14 deaths since 1994, the college did lay blame on substandard care. Three of the 14 doctors had their licences revoked, while the rest were handed reprimands or given short licence suspensions.

On Aug. 20, 1993, Tom and Patricia Bain buried their only son, 14-year-old Jonathan. At the time, the Mississauga couple were told his death was caused by a viral infection.

The family contends that tests later revealed prescribed drugs caused the teenager's death. They complained to the college that Oakville physician Anthony Laws covered it up.

Throughout the process, Laws continued to practise with a clean record.

College documents show Laws first prescribed the drug Cylert, which has been linked to liver problems, to treat Jonathan's Attention Deficit Disorder (ADD) in 1992.

In July, 1993, the boy was admitted to hospital with severe liver problems. He died after undergoing two liver transplants.

Following the teen's death, tissue tests revealed Cylert was the cause of Jonathan's liver failure, according to the family.

The Bains complained to the college that Laws ``never told us of the risk in advance, he never monitored our son when he was on the drug, he never ordered blood tests despite the risks.''

The Bains are upset that Laws prescribed Ritalin, another ADD drug, along with Cylert.

``He was experimenting with our son,'' Tom Bain told The Star.

But at the hearing, medical competence wasn't the focus. The panel was more concerned about Laws' conduct after Jonathan's death, ruling that he falsified letters to the Bains' family doctor and entries in Jonathan's chart and failed to warn them about the risks of liver failure.

``Our complaint was never dealt with because our complaint was never for forgery, falsification or perjury,'' Bain says. ``It was for the medical aspect.''

In June, 1999, Laws was found guilty of professional misconduct and handed a six-month suspension, reduced to three for meeting conditions such as seeking psychiatric treatment and upgrading his skills.

`He should have been revoked.'

- Tom Bain
referring to the family's complaint
about Dr. Anthony Laws

`(Laws) felt the disciplinary decision was fair and he didn't have any complaint about the process.'

- Tom Curry
Laws' lawyer

``He should have been revoked,'' says Bain. ``The system failed us.''

Tom Curry, the Toronto lawyer representing Laws, said his client ``felt the disciplinary decision was fair and he didn't have any complaint about the process.''

Like all complainants, the Bains had no official standing before the college, acting merely as witnesses in the case.

College lawyers, much like crown attorneys, act on behalf of the system, not the individual. Complainants cannot call witnesses or ask questions.

If complainants want their own legal advice, they must pay for it themselves.

On the other side of the room, money is no object for doctors. The Canadian Medical Protective Association (CMPA), which represents more than 90 per cent of doctors in Ontario, picks up the tab for top legal representation.

Doctors, in turn, are reimbursed with public money for most of the insurance premiums they pay. This year, it is expected the province will pay just under $70 million out of total malpractice premiums of about $100 million.

``I've walked out of college hearings thinking physicians are protected far more than they deserve,'' says Toronto lawyer Barry Swadron, who has represented several clients in complaints against physicians.

``Doctors stick together. If a fight is involved, the odds against the victim are astronomical.''

College president Gerace dismisses the claims of a power imbalance between doctors and complainants, saying the system is designed to protect the public interest.

``The analogy is the criminal system. If someone gets drunk and kills my child, I want to be a party to that but I'm not. The prosecutor is there to protect the public interest,'' he says.

``We're there to ensure that either a physician is found to be okay or is found to be wanting and that the correct steps are taken.''

Investigative reporter Robert Cribb can be reached at: rcribb@thestar.ca

FILE:COPY

Iatrogenic Stemetil Poisoning...!

2009-04-24T14:45:00.000-07:00

Iatrogenic Stemetil Poisoning...!

The evidence speaks when victims cannot

"Truth cannot live on a diet of secrets
withering within entangled lies"
H. Michael Sweeney

Introduction

Just as a forensic scientist can turn the smallest clue into a material witness, one can look to the medical record for clues that will bear witness to what went wrong and why. Neuroleptics (antipsychotics) are among the most dangerous medications ever used in medicine, and Stemetil is no exception. They are presently prescribed haphazardly by psychiatrists and general practitioners alike.
In simple words, they are brain damaging chemicals.

Prelininary Findings

All drugs are poisons and all poisons are (potentially) drugs: drug side effects rangie from the annoying to the lethal. There is also a physical tolerance and withdrawal syndrome (excitement seizures) , and a panic factor (iatrogenic) which can kill you.

Objective: To make a greater body of awareness of definitive evidence available from all sources of information relating to the drug Stemetil that shows presence or absence of the characteristic drug signature (pattern of injury) , especially where injury is believed to be doctor caused.

Stemetil, also known as prochlorperazine is used for the treatment of the symptoms of psychotic disorders such as excessive anxiety, tension, confusion, delusions, and agitation. It works by affecting the chemical balance in the brain. It also can be used to treat or prevent nausea and vomiting caused by the use of certain medications (e.g., cancer treatments) or motion sickness.

Target focal point: Drug signature analysis/profiling of key drug induced toxicities in liver, kidney and heart tissue associated with phenothiazine type drugs. By using biomarkers of drug pathology and mechanisms from drug signature (pattern recognition) and multi-source library profiles I will demonstrate and show publicly that use of Stemetil for control of nausea can be an iatrogenic drug induced nightmare of unprecedented magnitude.

Biomarkers are genetic indicators that have been shown to be correlative with normal biologic processes, pathologic processes or pharmacologic responses to theraputic intervention.

Probable Valid Biomarkers are currently defined as those that have been measured in an analytical test system with well-established performance characteristics and for which there is a scientific framework or body of evidence that appears to elucidate the physiologic, toxicologic, pharmacologic or clinical significance of the test results.

Known Valid Biomarkers are currently defined as those that have been measured in an analytical test system with well-established performance characteristics and for which there is widespread agreement in the medical or scientific community about the physiologic, toxocologic, pharmacologic or clinical significance of the results.

The liver plays a major role in the biotransformation of drugs and toxins and thereby becomes a major target for drug-induced injury. But what about other vital organs?

Stemetil (prochlorperazine) is a high risk antipsychotic-antiemetic drug to be used with caution, and according to manufacturer's directives. The drug Stemetil is produced in Canada by Aventis Pharma: formed by the merger of Rhone-Poulenc Rorer and Hoechst Marion Roussel.

Stemetil is widely distributed into body tissues and fluids. It suppresses activity in the trigger zones of the vomiting center by paralyzing the gastrointestinal tract that governs the vomiting reflex. Stemetil undergoes metabolism in the gastric mucosa and on first pass through the liver where it enters the enterohepatic circulation and is excreted chiefly in the feces.

Each drug tends to have a signature, meaning a typical pattern of injury. In the case of phenothiazine type drugs, this pattern of injury closely mimics viral hepatitis, clinically, biochemically and histologically. Patients typically have a mixed cholestatic-hepatocellular picture as seen in both bilirubin and aminotransferase levels. But don't be fooled by normal liver enzyme levels. This is a typical feature of edited lab records by callous physicians to conceal evidence of iatrogenic injury. Turn the focus to mechanisms from drug-signature with emphasis on pattern recognition. Make a detailed list of all signs and symptoms for comparative analysis. If typical pattern of injury is present but the liver function findings tend to elucidate or do not support the facts you can be reasonably certain that the records have been messed with. This of course requires a high index of suspicion, but a simple test to confirm is to detail one topic at a time, the blood pressure, for example. List everything in chronological order according to time of event from all sources of the record. If one record documents a certain blood pressure at a certain time, and another record or chart documents something different for the very same time you know the record has been altered. Then go on to the heart rate, etc. Leave no stone unturned. By repeating the procedure from different sources you can turn the smallest clue into a material witness.

If the clinical presentation is consistent with a hepatitis-like picture and/or cholestatic reaction, the findings are compatible with drug signature. There are a variety of clinical syndromes which may in some situations overlap. Severe infection is no exception. Always be on the alert for common denominators.

Stemetil (prochlorperazine ) poisoning is marked by oversedation, respiratory depression and hypotension. Symptoms of overdosage include sedation, loss of consciousness or coma , hypotension, tachycardia, EEG changes, ventricylar arrhythmias, and hypothermia or temperature dysregulation which may mimic an afebrile state. Although many of these signs and symptoms may overlap with certain conditions there are other ways of tracing pattern of injury, such as by systematic reviewing of all relevant literature and comparing it with the events documented on a patient's medical record. You'll be surprised at what you can uncover.

Stemetil is highly bound to proteins in blood (91-99%) and has a duration of activity from 4 to 6 hours. One caveat is that the drug has a half life that can potentiate or even interact with concurrent medications, such as analgesics, for example.

WARNING

Blood borne infection or meningocci in the blood lyse easily. It seems logical to assume that Stemetil would be contraindicated to infection for this reason.

Phenothiazines can potentiate and mask the signs and symptoms of overdosage of other drugs or underlying conditions, ie. organic brain syndrome, etc.. Thus patients can have exascerbation of their underlying or pre-existing conditions. The adverse effects of phenothiazines can affect all organ systems, and Stemetil is no exception; eyes, nose, and throat; internal organs such as liver, stomach, intestines, cardiovascular system, sexual organs, and especially the brain.

A great many patients suffer disastrous consequences, such as neuroleptic malignant syndrome, cardiovascular crisis, seizures, heatstroke, and sudden unexplained death, all of which can be attributed to medical stupidity and negligent use of phenothiazines, and their derivatives.

Phenothiazines type drugs are among the most dangerous medications ever used in medicine. Don't be fooled into believing that these drugs are actually treating a disease. They are suppressing overall brain and vital organ function, while creating diseases, and exascerbating illness, resulting in catastrophic decline of patient condition. Indications are primarily in the management of psychotic or mental disorders; also used to subdue patients - a chemical straightjacket. Reference: Psychiatric Insanity
.
The antiemetic action of Stemetil may actually mask the signs and symptoms of drug overdosage and may obscure the diagnosis and treatment of other conditions, ie. intestinal obstruction. How could they even think to market such a dangerous drug? An how could Health Canada even permit it?

Toxic doses of Stemetil can lead to changes in the blood-brain barrier (BBB), allowing an infectious agent to gain entry to the brain and produce lethal central nervous system (CNS = brain and spinal cord) infection. The scientific literature describe two bacterial factors specific to the meningitis pathogen that thwart the normal protective role of the blood-brain barrier, leading to serious infection. Further, sugar solution in IV creates gaps in the blood-brain barrier allowing chemicals to enter. Infected material can block the blood vessels to the brain, but Stemetil can help shuttle it directly into the brain and CNS.

Stemetil (prochlorperazine) enhances permeability of the blood-brain barrier and crosses the blood-brain barrier, breaching BBB integrity, thus allowing penetration of normally excluded agents, taking with it blood toxins and poisons in the blood-stream - which then triggers an inflamatory response. Bacteria invade the cerebrospinal fluid (CSF) by crossing the blood-brain barrier through a complex series of events, beginning with attachment of bacterial fibrils to the brain microvascular endothelial cells. Once in the CFS, bacteria multiply rapidly due to the absence of local defences where they further degrade the BBB and exascerbate tissue damage resulting in permanent brain injury.

Brain injury caused by drug induced meningitis related inflamation, edema, reduced blood flow (ischemia), or endogenous toxins can lead to neurologic sequalae, including deafness, mental retardation, motor dysfunction, and (secondary) epilepsy.

Stemetil is contraindicated to emesis in altered states of consciousness, coma, trauma, toxic syndromes, in the presence of blood dyscrasias, central respiratory failure, in the presence of circulatory collapse, in severely depressed patients, liver and renal insufficiency, intestinal obstruction, in patients with cardiovascular disorders, organic brain syndrome, or a history of hypersensitivity to phenothiazine derivatives.

Phenothiazines suppress sweating, causing central nervous system impairment which can infer temperature disregulation.

Stemetil contains a cardiotoxic metabolyte (having an injurious effect on the heart) which can lead to toxic myocarditis or drug induced cardiomyopathy, and subsequent heart failure. Current use of antipsychotic medications is associated with a more that tripled risk of Sudden Cardiac Death.

Stemetil is a high risk antipsychotic anti-emetic drug. Indications are primarily in the management of psychotic disorders. Stemetil (prochlorperazine) is the most potent in suppressing the reflex of nausea and vomiting. But that's not all it suppresses. Sudden deaths have occurred in hospitalized patients using this type of drug in which iatrogenic causes are suspect at the grass root level. In some cases, the deaths were attributed to cardiac arrest, while in others the cause appeared to be asphyxia due to failure of the cough reflex, the result of phenothiazine induced muscle paralysis.

Antipsychotic drugs may cause blood clots, researchers report extending previous findings. Prochlorperazine has been reported to cause heart attack, shock, stroke, and GI bleeding. Phenothiazine type drugs have even been reported to trigger diabetes in patients with no previous history of diabetes.. Neuroleptic agent toxicity in an already sedated patient can result in tachycardia, hyperthermia, urinary retension ileus, mydriasis (pupil dilation), hypoglycemia (becomes apparent as blood toxicity increases) toxic psychosis, and can even cause direct injury to the heart.

Drug Induced Dystonia is defined as a movement disorder characterized by sustained muscle contractions. freequently causing twisting and repetitive movements or abnormal postures that can result in distorted postures.It is a charcteristic feature with phenothiazine type reactions.

Dystonic reactions are thus acute spasms of muscle groups that can lead to a fixed upward gaze... painful dystonic reactions can be quite frightening to patients. These reactions typically occur soon after initiation of an antipsychotic drug. When that happens any negligence of the patient's throat secretions may lead to aspiration of vomitus or gastric content and a vicious cycle begins, which if not broken will lead to death.

Incontinence suggests loss of bladder control, or bladder paralysis, and is a very serious side effect of Stemetil, and possibly first sign of a drug reaction.

Compare: Rapid-Onset Dystonia Parkinsonism (RDP), a rare form of dystonia also characterized by dystonic spasms in the limbs, with prominant involvement involvement of the speach and swallowing muscles. Symptoms can be sudden, occuring over hours. Suppression of the gag reflex due to swallowing difficulty attributed to muscle paralysis can result in aspiration of vomitus or gastric content and ultimately, in asphyxia.

Tardive dyskinesia (TD) is also characterized by involuntary movements of the arms and legs. TD is potentially irreversable. Spontaneous dyskinesia has occured as abnormal involuntary movements in persons who have never been previously exposed to antipsychotic medications. Opisthotonus Iis characterized by spasms of the body where the head and heels are bent backward and the body is bowed forward. It occurs with drug reactions triggered by IV infusions with drugs such as Stemetil. Compare Idiopathic spasmodic torticollis, a focal dystonia associated with neuroleptic drug toxicity.

Stemetil changes a variety of chemicals in the brain. The adverse effects of Stemetil can affect all organ systems and may be attributed either to the drugÕs effects on the central autonomic nervous system, or to hypersensitivity reactions to the drug-reaction. Increased sedation is a serious side effect of this type of agent.

Symptoms of overdosage include central nervous system (CNS) depression which may vary from simple lethargy to coma. Symptoms of overdosage may be more severe than usual side effects, or two or more of the following may occur together: Falling levels of consciousness or coma; convulsions (seizures); fast or slow heartbeat; large fixed dilated pupils (also associated with shift of brain tissue and transtentorial herniation); palor; dry skin; weak pulse; low blood pressure; tachycardia; trouble in urinating; twitching or jerking movements; and vomiting. Other possible manifestations include paralytic ileus, hepatic dysfunction, renal dysfunction, gastrointestinal disturbances, respiratory depression (breathing difficulty), cardiac arrhythmias (heart failure), skin rashes or fixed drug eruption, dry eyes, ocular gyric crisis (eyes rolling back into their orbits); and simptoms resembling Parkinson's disease .

Caveat: Anticholinergic agents such as Gravol, an antihistamine, in combination with Stenetil can inhibit gastrointestinal motility which can exascerbate gastrointestinal problems, such as constipation or bowel obstruction . When the bowel stops working the body gets toxic. The effects of anticholinergic drugs such as Gravol may be potentiated by neuroleptic type anti-emetics, such as Stemetil. Use of neuroleptics in combination with anticholinergic agents may also increase the risk of tardive dyskinesia. The effects of opiate ANALGESICS (morphine) may also be potentiated similarly with concomitant or concurrent Stemetil use. Moderate to severe drug-drug interaction with Stemetil (prochlorperazine) and Dimenhydrinate (Gravol) with additive effects when used in combination or concurrently is reported in the literature. Search: Anticholinergic Syndrome. Compare Antichollinergic Toxidrome.

Life threatening: Uncontrolled mucscle twitching characterized by stiffness or spasms of the muscles, swirling tongue movements or swelling of the tongue, difficulty swallowing are all common features of the Neuroleptic Malignant Syndrome (NMS). Neuroleptic Malignant Syndrome is characterized by alterations in consciousness, and autonomic instability, such as tachycardia, hypertension or hypotension.

Stemetil poisoning is marked by oversedation, respiratory depression and hypotension. All phenothiazines can potentiate and mask the signs and symptoms of overdosage of other drugs or underlying conditions, ie. hypotension, etc. Thus patients can have exascerbation of their underlying disorders.

Phenothiazines that are clearly contraindicated in a wide variety of medical conditions have no effect on systemic infection, EXCEPT TO EXACERBATE PRIMARY or UNDERLYING CONDITIONS due to a whole array of clinical insults, any or all of which can trigger a procoagulation response which can ultimately lead to coma, circulatory collapse, and DEATH., which is also a major cause of unexplained sudden dealths in hospitalized patients, more so than most ostrich-like physicians care to admit.

The neuroleptics or antipsychotics are the most frequently prescribed drugs in mental hospitals, and are widely used in board-and-care homes, nursing homes, institutions for people with mental retardation, children's facilities, and prisons. They also are given to millions of patients in public clinics and to hundreds of thousands in private psychiatric offices AND NOW IN OUR HOSPITALS, PRESUMABLY FOR CONTROL OF NAUSEA. Often they are prescribed for anxiety, sleep problems, and other difficulties in a manner that runs contrary to the usual recommendations. And too often, they are administered to children with behavior problems, even children who are living at home and going to school. Rather than treating a disease, the neuroleptics create a disease. The neuroleptic drugs are chemical lobotomizing agents with no specific therapeutic effect on any symptoms or problems. Their main impact is to blunt and subdue the individual. They also physically paralyze the body, rendering the individual less able to react or to move. They produce a chemical lobotomy and a chemical straitjacket. The drugs are also the cause of brain damage that afflicts up to half or more of long-term patients. The original ones, including Thorazine and Mellaril, are called phenothiazines.

Chemical Lobotomy:

While the neuroleptics are toxic to most brain functions, disrupting nearly all of them, they have an especially well-documented impact on the dopamine neurotransmitter system. Dopamine neurotransmitters provide the major nerve pathways from the deeper brain to the frontal lobes and limbic system--the very same areas struck by surgical lobotomy. Most psychosurgery cuts the nerve connections to and from the frontal lobes and limbic system; chemical lobotomy largely interdicts the nerve connections to the same regions. Either way, coming or going, it's a lobotomy effect. Clinically, the drugs produce a lobotomy and neurologically the drugs produce a lobotomy. Starting from two main trunks deep in the brain, the dopamine nerves spread out like the branches of a tree, reaching into the emotion-regulating limbic system and frontal lobes. This dopamine tree is shut down by the neuroleptics.

Tardive dyskinesia is a movement disorder, frequently caused by neuroleptics drugs that can afflict any of the voluntary muscles, from the eyelids, tongue, larynx, and diaphragm to the neck, arms, legs, and torso. On rare occasions it can occur after a few weeks or months, but usually it strikes the individual after six months to two years of treatment. Some psychiatrists try to blame the neurological disorder on schizophrenia rather than on the drugs. It manifests as uncontrollable twitches, spasms, or writhing movements. Any of the neuroleptics can cause tardive dyskinesia.

We've all heard of the Chemical Straightjacket. To be put in a chemical straightjacket can be terrifying.

In summary, the neuroleptic drugs are chemical lobotomizing agents with no specific therapeutic effect on any symptoms or problems. Their main impact is to blunt and subdue the individual.n be terrifyingthey also physically paralyze the body, rendering the individual less able to react or to move. Thus they produce a chemical lobotomy and a chemical straitjacket

CONTRAINDICATIONS/PRECAUTIONS: All phenothiazines, including prochlorperazine can cause neuromuscular reactions, particularly dystonias.

Prochlorperazine can worsen conditions in patients with organic or traumatic brain damage. This is a relative contraindication, and the exact cause of this phenomenon is unknown. Patients with underlying temperature dysregulation can have this condition amplified by prochlorperazine. In patients with underlying tardive dyskinesia, prochlorperazine can worsen the condition in the long term. An alternative antiemetic or anProchlorperazine can cause hypotension, so patients with cardiovascular disorders could have exacerbation of their conditions if administered prochlorperazine. tipsychotic should be considered. Concomitant administration of prochlorperazine and other anticholinergics, including atropine, monoamine oxidase inhibitors, phenothiazines, antihistamines, antidepressants, meperidine, and antiparkinsonian agents, can cause additive adverse effects such as oversedation, paralytic ileus, and severe constipation.

Stemetil is widely distributed into body tissues and crosses the blood-brain barrier. The drug STEMETIL has a history of adverse reactions resulting in -induced hypoxia with irreversible brain damage due to oxygen starvation of the brain, including "sudden, unexpected and unexplained s" in hospitalized patients.

Parkinsn's List DataBase prochlorperazine / Compazine, ... describes Stemetil as a HIGH RISK antipsychotic-antiemetic with a history of adverse effects most commonly related to the "high potency" neuroleptic . Prochlorperazine (Canadian generic name for the same medication. Stemetil is hepatically metabolized (well absorbeed into the gastrointestinal tract, with high concentrations into the liver and spleen).

The concept of a systemic inflamatory response syndrome expresses the notion that the body responds in certain ways to a variety of insults and negligent use of Stemetil is the ultimate clinical insult, physically, and neurologically. All virtues of phenothiazines can potentiate and exascerbate similar or like-virtues of other medications known to cause respiratory depression. In frail patients, as respiratory rate decreases, the patient becomes increasingly sedated. Morphine is one drug that can lead to respiratory depression. Concomitant or concurrent use of Stemetil can result in exacerbation of this condition.

Caveat: Prochlorperazine intoxication or poisoning can also cause deep physiologic depression that resembles and can mimic brain death. SEARCH: Brain Damaging Neuroleptic Drugs.

The neuromuscular blocking effects of neuroleptic drugs are many and include nerve paralysis including neuronitis of spinal nerves. There is also a panic factor which can result in heart failure and kill you. Remember: The heart is a muscle, as are other vital organs.

Compare drug induced Rhabdomyolysis in which drug-toxicity involves vital organs : kidney, liver, gastrointestinal tract and CNS , with skeletal muscles usually being less readily affected. Drug induced Rhabdomyolysis is the breakdown of muscle fibers resulting in the release of muscle fiber content into the circulation. Rhabdomyolysis from any cause leads to inadequate blood-perfusion and subsequent renal failure - cardiogenic shock or cardiorespiratory arrest may occur exceptionally.

The bald truth is that neuroleptic drugs are chemical lobotomizing agents with no specific therapeutic effect on any symptoms or regimens, and Stemetil is no exception. Neuroleptics (antipsychotics) inhibit dopamine nerve transmission in the frontal lobes and in the emotion-regulating limbic system of the brain. This inhibition is no different than surgical lobotomy. It is chemical lobotomy. Their main impact is to blunt and subdue the individual.. They also physically paralyze the body, rendering the individual less able to react or to move. Thus they act as a chemical lobotomy and a chemical straightjacket . In some cases these types of drug can physically paralyze the intestines, vital organs, including the inspiratory muscles of the diaphragm (diaphragmatic paralysis) literally smothering the patient, resulting in overwhelming exertional fatigue or stress , the panic fright factor response to chemical straightjacket, followed by a seemingly slow and painful death due cardiorespiratory arrest in the presence of circulatory collapse. Compare with the "locked-in state", a condition in which a person is conscious and able to think, but is severely paralyzed due to nerve paralysis. In my opinion which is widely shared, neuroleptic drugs should be banned.

Put yourself in a patient's position of total or near total paralysis with most of your senses blunted, unable to move, speak or even open your eyes due to a severely paralyzed motor function - you try relentlessly to free yourseld until you become so overwhelmed by exhaustion, fright and panic that your heart begins to gives out.

Phenothiazine derivatives are multipotent receptor blockers that give rise to a large number of pharmacological responses. The receptor-mediated responses can be ascribed to either their effect on central neurotransmission or to their influences on neuronal terminal junctions in peripheral tissues. Centrally, phenothiazines are postsynaptic antagonist and they inhibit catecholamine re-uptake at neuronal terminals. The antagonism in the basal ganglia results in neurolepsis while antipsychotic effects arise from action in the limbic system and hormonal side effect from activity in the hyphothalmus.

Peripheral responses to these drugs are as a result of predominantly an adrenegis blockade. They are often complex in their pharmacological interpretation as neuronal reuptake, which reduces catechlomine-medicated responses is inhibited at the same time. Peripheral and central cholinergic antihistaminic and antiserotonin effects also contribute to the peripheral manifestation.

CAVEAT: Peripheral nerve disorders that cause paralysis can result. Examples of peripheral nerve disorders include Guillain Barre´ Strohl Syndrome, Chronic Inflammatory Demyelinating Polyneuropathy (CIPD), and Diabetic Neuropathies.

Stemetil Can Kill You...!

In order for drug to attenuate the effects of an agonist it should not be contraindicated. Contraindicated medications may backfire dramatically, provoking opposite effect for a number of reasons, such as potentially lethal incorrect dosages, especially with medications that are contraindicated and dangerous v. the failure adequately to monitor, v. failure to perform an appropriate medical screening, v. failure to determine the etiology of disorders. The term "backfire" means "to have the reverse of the desired or expected effect" - usually follows a course of inappropriate treatment due to misdiagnosis.

Toxicity of antipsychotic medications may be increased by co-ingestion of other agents, particularly drugs with similar metabolic pathways. Sedative effect may be apparent before antidepressant effect is noted. Adverse effects of medications include somnolence and sleep attacks. Sleep attacks are indistinguishable from drug induced somnolence and excessive sleepiness. Sleep attacks are defined as events of overwhelming sleepiness that occur without warning. Sleep attacks in patients taking dopamine agonists: is reported in PubMed. Symptoms that make up the tetrad of narcolepsy: Sleep attacks that last with increased sedative effects - drugs cause sleep attacks. Combining drugs such as prochlorperazine and opioids, may potentiate adverse effects, such as sedation and respiratory depression.

Neuroleptic malignant syndrome (NMS) is caused almost exclusively by the blocking of dopamine receptors with antipsychotic medications, including all types of neuroleptics (typical and atypical antipsychotic drugs).The higher the dosage of the antipsychotic, the more common the occurrence. Rapid and large increases in dosage can also trigger the development of NMS. NMS is a potentially fatal complication of therapy with antipsychotic medications.

Stemetil® is no longer being manufactured for sale in Canada.

There is growing concern in Ontario that far too many doctors are prescribing "contraindicated" medications with utter disregard for patient safety, and Stemetil is no exception. This is a very bad trend. Cases involving injury or death have been reported as allergic reactions. The exact number of Stemetil related injury and/or deaths is unknown because less than 10% of drug outcomes are reported. Many of the cases are undoubtedly the result of imprudence on the part of incompetent or negligent physicians. Medical stupidity may predispose. In my opinion, doctors who prescribe or order "contraindicated" drugs for their patients are guilty of criminal negligence. They should be divested of their right to practice medicine, fined heavily, and jailed. In cases where death results from failure to at least comply with the manufacturer's directives (WARNINGS), the exact same penalties that apply to "negligence causing death" should be handed down by indictment. The law applies equally to everyone, physicians are no exception.

Case in point: A hunter may be licenced to carry guns "for the pupose for which they are intended", namely hunting game. He has no right to discharge the guns "BLINDLY" with utter disregard for public safety. There's a similar case in point that could also be made for "reckless" licensed drivers who don't obey rules of the road and "ignore" WARNING signs, etc., etc., as a rule of thumb. Physicians who elect to act with "wanton and reckless disregard for human life" should be treated NO different. There's a barrel full of bad doctors out there that need to be shown the error of their ways, otherwise nothing changes.

A noble is to right wrongs and smite injustice whenever and wherever they encounter it for the good of those people which have been entrusted into their care. Turning a "blind-eye" to such negligence shows little or no concern for public safety. This is not unusual for a government backscratching drug trade bent on the path of corruption where grand corruption is rampant at the top level of government - a common feature of the McGuinty government in Ontario (typical of a constituency severely lacking in social and moral values), whose only values evolve around the almighty dollar, and protecting each other from being PROSECUTED. It seems clear that we are living in a nation of cowards and shirkers.

PSYCHIATRIC DRUGS: Cure or Quackery?

"Psychiatrists and Neuroleptic drugs are a regular
nightmare. Deleterious torturing techniques must dance in their heads".
M. Svoboda.

Crimefest - Eh? Fake Doctors Notes?

2009-04-08T22:02:00.000-07:00

"Eh? Fake Doctors Notes?"

Don't Get SCAMMED by Junk Fake Doctor Notes.

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Popping Super Growth Pills: The Case for Cognitive Enhancement?

2009-03-30T15:56:00.000-07:00

Popping Super Growth Pills: The Case for Cognitive Enhancement?

TIME Magazine - The Deadly Pills

www.time.com/time/magazine/article/0,9171,879362,00.html

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Code 0: Patient Status Code 0

2009-03-23T15:52:00.000-07:00

Publication In Good Faith For Redress Of Wrong

Status Code 0

Code 0: No Code = No Care

“Do Not Resuscitate" (DNR) means that a patient who suffers sudden cardiac or respiratory arrest will not receive Cardiopulmonary Resuscitation (CPR). DNR is also referred to as a "No Code"

The Arlene Berry Death Coverup

In a letter to the College of Physicians and Surgeons of Ontario dated November 28, 2000, Dr. Jordan writes "discussed the situation with family members and a decision was made to intubate Ms. Berry", while the Ambulance Call Report seen at N-7 documents an unsigned "Status Code 0", or endorsement of a No Code by proxy. This is an order akin to a 'DNR' denying medical intervention.

Patient condition "Status Code Zero" taken from CMAJ articles, is used to describe a "dead" patient condition. The Yale Law Journal, Vol. 93, No. 2 (Dec., 1983), pp. 362-383, criticizes questionable hospital policies "that resort to third party adjudications in No-Code decision making", and provides insight into the assigning of a "no-code order" instructing personnel "not to attempt resuscitation", as in this case.

In a hospital, an order to withhold resuscitation is commonly called a "no code" (code 0). Only a doctor can write an order for a patient to be a "no code."

A full code is the most extensive course of treatment. It communicates to the physicians that all measures should be taken to keep the patient alive. The patient is only considered safe when his/her code status is full code.

Although it is clear that Arlene Berry was transferred to Sudbury with ventillatory support, and although Drs. Jordan and Spiller were aware of the need for emergency care and life support, after ordering it, they canceled it, using the secretive no code endorsement as a pretext for evoking a declaration of death and in fact waited for the patient's death.

Looking over the chart it is clear that obtaining a 'no code' status in the face of immune mediated adversities by reason of Dr. Jordan's and Dr. Spiller's failure to recognise and treat an emergency situation accordingly, and by reason of Dr. Jordan's failure to attend in a timely manner was the next essential step for these two negligent physicians in executing Arlene Berry's death in order to avoid liability issues. Turn off the respirator and in the natural course of affairs the patient dies from lack of oxygen.

Within a few hours following her transfer from the Kirkland and District Hospital to the Sudbury Regional Hospital Arlene Berry was declared as having met with 'brain death criteria' , while under the care of Drs. Sauve and Adegbite. Her remains were kept in Sudbury for several days prior to being returned to Kirkland Lake.

Withholding life sustaining treatment from an undiagnosed patient with concurrent hyperglycemia, hypokalemia and electrolyte abnormalities in combination with a severely paralysed motor function and who is under the influence of sedative hypnotic and tranquilizing agents is of questionable legality. Death results from respiratory paralysis and subsequent asphyxiation.

Passive euthenasia involves an allowing of "nature to take its course", while active euthenasia consists of killing someone (to do acts causing death), or by choosing not to act is also an act, which determines the course and the outcome of events.

To practice euthenasia by withdrawing life support to a critically ill patient is a medical homicide (to kill or destroy by preventing access of air or oxygen).

When life support is discontinued, death results from respiratory paralysis and subsequent asphyxiation. In the strict medico-legal sense it signifies asphyxia, caused by lack of oxygen or obstruction of the breathing passages.

The clinical diagnosis of brain death in this case was made in the presence of metabolic derangements and endocrine abnormalities and constitutes an act of wanton and reckless disregard for human life.

There have been many challenges to the several concepts of "brain death" and the means of their diagnosis worldwide (vide infra). Indeed, it seems that there is now an emerging consensus that "brain death" diagnosed by any of the protocols in current use worldwide is "not death."

CJNS-Guidelines for the Diagnosis of Brain Death

Brain Death:Inconsistencies

An act of wanton and reckless disregard for human life is an act of criminal negligence, in this case, causing death. In my opinion, Arlene Berry's death was premeditated, as evidenced by the physician's documented status "Code 0", and Dr. McLellan knowingly returned a false finding for which he remains to be held to account. From the facts of this case it seems clear that this moral compass isn't such a straight arrow after all.

The Criminal Code of Canada regards euthanasia, whether passive or active, as culpable homicide, or murder: A culpable homicide is defined as murder "where the person who causes the death of a human being means to cause his death" (s. 229, Criminal Code of Canada). Unless the law has changed in recent years, euthanasia carries a fixed, minimum penalty of 10 years in prison. euthanasia carries a fixed, minimum penalty of 10 years in prison.

Don't Kill Me Doctor:

2009-03-17T07:51:00.000-07:00

Wash Your Hands!

It's hard to believe. The chances are only 50-50 that the doctor treating you in the hospital, even when performing your surgery, has washed his hands. The odds are the same as flipping a coin. Actually it's worse than that. According to researchers, hand-washing compliance rates at hospitals are generally less than 50 percent.

Methicillin-resistant staph aureus, or MRSA, is the strain of a once-innocuous staph infection that has become invulnerable to first-line antibiotics and kills more people every year in Canada and the U.S. than the AIDS virus and in the majority of cases is contracted in hospital settings.

Need A Hand?

IATROGENIC

2009-03-15T19:27:00.000-07:00

Our Health Care System is the Leading Cause of Death. Doctors are in fact the LEADING cause of or preventable death in this country. Not heart disease, not cancer, not cigarettes--doctors. In all fairness, doctors themselves are not always to blame for all of this. The entire modern health care system, however, is to blame for allowing, even promoting, so many unnecessary procedures, drugs and mishaps and for allowing bad doctors to go unchecked.

Drugs and doctors kill far more people annually than they save, and that's a fact. And the older you get, especially if there's basically "nothing wrong," the more likely that you'll "check out" because of a medical error or drug reaction.

Doctors kill so many people that medical grammarians gave the doctor-based killing a name: IATROGENIC.

Tip: Share what you read.

More Doctors Smoke Camels Than Any Other Cigarette

2009-03-15T18:56:00.000-07:00

Get The FACTS!

Alert On MD's

Tip: Share what you read.

New Cigarette WARNING!

2009-03-15T18:49:00.000-07:00

WARNING:

Tip: Share what you read.

Cannabis Canada!

2009-03-15T18:02:00.000-07:00

Medical Marijuana is Legal in Canada.

It is not illegal to grow cannabis in Canada as long as you have a license issued to you by the federal Minister of Health.

Get the FACTS

Canada Medical Marihuana

M.D. really stands for "Medical Dolt"!

2009-03-14T22:14:00.000-07:00

We struggle today, as a culture, to get over the idea that M.D. really stands for "Medical Dolt" (rather than "medical doctor"), because most doctors believe they are a god, having the power of life or death over others. For many physicians, such as the following, the title is well deseved.

RateMDs.com - Dr. Edward Henry Jordan Doctor Ratings

EH Jordan

Edward H. Jordan is a general practitioner with a known propensity for error on staff at the Kirkland and District Hospital, a hospital controlled primarily by ineffectual overseers and poachers of patients rights, where the order of the day is an ogre of uncivilized behavior geared to its own self-serving interests, where doctor unavailability nurtures patient habit of seeking out the ER, and tenure of welfare cases is seen as a deterrent to good medical care. Jordan was born in 1962 and claims to be a graduate of the Memorial University of Newfoundland, graduating in 1988. It is not known whether he attended per se, or graduated by correspondence. A search of the student yearbook for 1988 failed to confirm his academic attendance.

His postgraduate training, according to a CPSO Doctor Search, includes what I take to be a 3-day specialty crash course in obstetrics and gynecology at the University of Toronto in September of 2000. Dr. Jordan is now a "clinical fellow" who fancies himself a specialist in that field. This would not be unusual; the obstetrician has long been considered a midwife masquerading as a specialist.

Like many other doctors, he suffers from undiagnosed metacognitive deficit disorder, a syndrome that spans a broad continuum of metacognitive incompetence or lack of capacity to distinguish accuracy from error. Not only do these people reach erroneous conclusions and make unfortunate choices, but their incompetence robs them of the metacognitive ability to realize it. Faulty reasoning or inability to pay attention may predispose. In his efforts to gain hands-on experience for academic completeness, he is indifferent to and ignorant of the near epidemic harm that accompanies him like an evil talisman. His shortcomings at the Kirkland and District Hospital not only include a litany of poor choice medications with
severely ill patients, but also an implacable pattern of bad habit and lack of concern and in particular, with respect to indigent female patients and young mothers-to-be resulting in increased infant mortality with induction of labour at his, rather than natures convenience, and myriad more.

A consensus of local opinion is that all of Dr. Jordan's patients are either inert, erroneously medicated, or dead. Further findings suggest that he perceives his patients as an intrusion, or commodity that is, unfortunately necessary to maintain his practice. Further, word on the street has it that a number of civil malpractice suits have passed his way on several ocassions. One might wish to look up Dr. Jordans name in the general division/civil actions of the provincial court house for malpractice claims and/or follow his death trail by seeking out the families of those who have died while under his care.

Healthcare Ontario

2009-03-14T20:31:00.000-07:00

The Arlene Berry Death Coverup

Sudbury Regional Hospital - Andrew Adegbite, MD - Topix

The Medical Record of Arlene Berry

DOCTORS' ETHICS?

2009-03-14T19:14:00.000-07:00

EXAMPLES OF THE LACK THEREOF

Ethics 101

The Iatrogenic Experience

2009-03-14T14:12:00.000-07:00

< Truth / Justice / Patient Safety >
It's a path

Disposing of patients to protect doctors

Silence Infers Enough!

2009-03-14T08:53:00.000-07:00

"Truth Cannot Live on a Diet of Secrets
WitheringWithin Entangled Lies" H.Michael Sweeney

Silence can be good or bad. In general, silence (ex silentio) refers to the claim that the absence of something demonstrates the proof of a proposition. Silence infers consent (tacit agreement, or acquiescence). Silence infers knowledge that he/she can’t (or wishes not to) reveal. Silence may infer assent to certain actions or inactions.

Silence is the opposite of safety

Silence can also infer a blame on mainstream medicine or media for pretending or ignoring. If an accused is silent, let the public record show that. Their silence infers that they may have done something "wrong". Silence infers arrogance, shame, impulse to hide, to run, to get away. That could definitely work in your favor when seeking the truth. However, this stunning silence suggests that newly berated doctors care more about their code of silence than they do about their own personal reputations.

BEWARE of extortion tactics!

Doctors try to silence negative reviews from patients

Silence may also infer a meeting of the minds to accomplish a common objective, an inference from which conspiracy theories are based.

When dealing with truth, liars and suppression of truth, or where a crime involves a conspiracy, or conspiracy to cover-up, accuse those guilty of the later freely. They (both those deliberately seeking to lead you astray, and those who are simply foolish or misguided thinkers) generally run for cover when thus illuminated.

As with all secret societies, the law of silence is absolute among doctors!" But if someone comes up with information that is not in keeping with the medical propaganda, and sticks to his guns ie. becomes a
whistleblower, just watch and see what happens.

If you're looking for a way to humiliate, you can always intimidate the experts into silence with a
"BIG MOUTH LAWYERS" tactic, for example.

If a person, be it a doctor, lawyer, or whatever has something to hide/lose this will definitely shut them all up:

Publication In Good Faith For Redress Of Wrong

Silence versus Patient Safety
Loyalty versus Patient Safety
The White Wall of Silence versus Patient Safety
Blacklisting Patients
Freedom of Speech for Patients
Medical Complaints - How to

* * * * * <Truth/Justice/ Patient Safety > * * * * *
It's a path

Visit: www.patient-safety.com

Report bad doctors

On Bias

2009-03-14T07:46:00.000-07:00

What the government and the medical profession doesn't want you to know.

Know that Ontario has a long and sad history of socially unacceptable medical corruption and criminal cover-up under shield of the College of Physicians and Surgeons of Ontario that has sought to institutionalize rip-offs with OHIP, and to keep sick people sick and their families oblivious to it and to shield bad doctors from medical procedure mistake(s) including civil and criminal accountability. So why not create a website under cloak of anonymity, call it RateMyMd.ca, for example, to fetter out what the government and medical profession consider "Scandalous, Frivolous or Vexatious", by means of medical control ploys designed to downplay by omission with the intended ability to influence ITS audience. Downplaying by omission is common since the basic selection/omission process necessarily omits what they don’t want the public to know, with cover-ups and criticisms suppressed and of course they will try to make you look absolutely stupid in the process. Bias is the single most serious problem when dealing with medical opinion and doctors are the worst offenders. There is no such thing as an unbiased medical professional and scientific misconduct goes far beyond discriminating against ethnic groups to include targeting the "have-nots" of society. All communication is limited, slanted, or biased to include or exclude relevant information. Anyone who has ever launched a formal complaint against a doctor for wrongdoing knows exactly what I am talking about.

Truth Suppression Techniques

Medical secrets revisited:

Medical Secrets
SexPatients

CANADA PPL Reporting bad Doctors?-ukcoalition.org

Only in Canada - Doctor ratings & reviews likely Censored!

2009-03-14T07:12:00.000-07:00

Rate Your Doctor - Canadian Doctor Ratings

This website holds itself out as a "free, non-biased, anonymous database" of Canadian doctor ...
Rating Dr. Edward Jordan Area Of Medicine: Family Doctor / General Practitioner
"My wife died unnecessarily as a direct result of this doctor's stupidity."

Doctors will try anything to silence negative reviews from patients.

Note: The all important information was obliterated following this posting.

"My wife developed "flu-like" symptoms within two weeks following radiation therapy, at the end of April of 2000. marked by loss of appetite, nausea, bilious vomiting and night sweats, suggestive of gastrointestinal illness. She complained of severe stomach pain that would come and go with bloody bowel movements ongoing for 3 days. She was given antibiotics for a “suspected UTI” and sent home. On the 4th week she became constipated and developed migraine headaches with increasing severity, weakness, difficulty ambulating with unsteadiness, drowsiness. She had “mild diffuse weakness” and “slurred” speech at presentation on May 23 rd of 2000. She appeared to be haggard, but was otherwise found to be “alert and oriented”. Examination revealed “no focal deficits”. She was afebrile. She was admitted to Kirkland and District Hospital for IV and Gravol. Among antibiotics, she was given 30 mg MS Contin (Morphine Sulphate) in the face of undiagnosed and undifferentiated emergent conditions associated with "abdominal pain" and bilious vomiting, followed by an order of 10 mg x 4 daily Stemetil (prochlorperazine)“for control of nausea”, after which she began to rapidly deteriorate with signs of fluid overload, including tachycardia, elevated blood pressure, dyspnea (labored breathing) and other signs of respiratory distress. In a letter to the College of Physicians and Surgeons of Ontario dated November 28, 2000, Dr. Jordan writes "discussed the situation with family members and a decision was made to intubate Ms. Berry", while the Ambulance Call Report seen at N-7 documents an unsigned "Status Code 0", or endorsement of a No Code by proxy. This is an order akin to a 'DNR' denying medical intervention. Dr. Jordan treated her over the telephone, unseen, while sitting at home watching TV. Looking over the chart it is clear that obtaining a 'no code' status in the face of immune mediated adversities by reason of his failure to attend, diagnose and treat accordingly was the next essential step in executing her death. This is an order denying medical intervention in emergency situations. The 'no code' as evidenced in this case by a "Nature Code 0" (no code = no care) was ordered by Dr. Jordan, without family knowledge or consent when the patient's condition began to rapidly deteriorate. To allow or hasten a patient's death to cover-up error or negligence is reprehensible. It is nothing short of murder since such an act or omission carries the intended consequence of the act or omission, hence, the mens rea or criminal intent. Her medical record had been altered and falsified in order to cover-up incompetence, gross medical negligence and medical stupidity. Only 41 years of age, my common-law wife died a tragic, horrific, and senseless death in the face of treatable and potentially reversible conditions, for which I hold Dr. Jordan criminally responsible. It took almost 8 years of private investigation and medical research to narrow the events down to overlapping variants of the Guillain Barre syndrome against a background of undiagnosed and untreated diabetic neuropathy."

When I complained in the feedback about the rating matter, here is what I received in my email:

mcrawford@searchstudio.net

" to me

show details 9:53 PM (27 minutes ago)

Rating Dr. Edward Jordan Area Of Medicine: Family Doctor / General Practitioner
Note: The all important information > <>>>“ s stupidity."

Know that bias always accompanies "errors and distortions". In the alternative, their system has an acquired "superbug" up its geekodomain.

That would not be unusual where a derailment of information "jumping the tracks" is the primary objective!

ratemymd.ca Estimated Worth $459.9 USD by websiteoutlook

Follow the story?

Dr. E.H. Jordan already has an extensive negative review profile which can be viewed at
RateMDs.com - Dr. Edward Henry Jordan Doctor Ratings
RateMDs.com | Doctor ratings and reviews

Status Code 0

2009-03-13T14:27:00.000-07:00

Code 0: No Code = No Care

The Arlene Berry Death Coverup

In a hospital, an order to withhold resuscitation is commonly called “no code" (code 0). Only a doctor can write an order for a patient to be “no code."

Passive euthenasia involves an allowing of "nature to take its course", while active euthenasia consists of killing someone (to do acts causing death), or by choosing not to act is also an act, which determines the course and the outcome of events.

To practice euthenasia by withdrawing life support to a critically ill patient is a medical homicide (to kill or destroy by preventing access of air or oxygen).

CJNS-Guidelines for the Diagnosis of Brain Death

Brain Death:Inconsistencies

The Criminal Code of Canada regards euthanasia, whether passive or active, as culpable homicide, or murder: A culpable homicide is defined as murder "where the person who causes the death of a human being means to cause his death" (s. 229, Criminal Code of Canada). Unless the law has changed in recent years, euthanasia carries a fixed, minimum penalty of 10 years in prison.

Rx for Injustice!

2009-03-12T16:09:00.000-07:00

WACKY MEDICAL DOCTORS- Think your doctor/MD is
just about God on Earth? Make 'em PAY!

Iatrogenic

R_x for Injustice - Can't beat 'em? Smear 'em!

The physician's failings, have now become the subject of sensitive political topic. They will no longer go unnoticed; the physician's too often slipshod diagnosis of illness; his faulty application of proven therapies; his creation of iatrogenic, or doctor caused illness and death; his wanton and reckless disregard for human life; his infatuation with the new and unproved; his serious shortcomings in applied pharmacology; his sometimes shocking excess of human experimentation and perversions of science followed by complications, pain, suffering, misery, disability; his comprehensive catalogue of errors of omissions and commissions; his often lifetaking deficiencies and heartwrenching butchery and debauchery, from malpractice to anesthesia death, medical stupidity and medical atrocities of all kinds will now be held out to public view.

If doctors don't understand ethics, its time to start teaching them...

Share on MySpace!

MedicalSecrets

2009-03-12T11:21:00.000-07:00

TheStar.com/doctors

Ontario has a long and sad history of socially unacceptable medical corruption and criminal coverup under shield of the College of Physicians and Surgeons of Ontario that has sought to institutionalize rip-offs with OHIP, and to keep sick people sick and their families oblivious to it, and to shield bad doctors from medical procedure mistake(s) and criminal accountability. Neither the government, nor the medical profession want to lift the manhole cover on this sewer of corruption, needless suffering, and unnecessary deaths.

Since fewer than 10 percent of medical mistakes are ever reported by hospital authorities, the patients, or their families are never told that the injuries were caused by the doctors. That means greater than 90 percent of doctor caused deaths go undiscovered, hence no disciplinary action is ever taken on any of the doctors involved in these incidents. It is also a measure of honesty and integrity in the medical profession, a profession that has its roots in occult formulas, medieval magic, and quackery of all kinds. The 1960's saw physicians in Canada go on strike and the mortality rate actually dropped.

The Toronto Star's courageous series on the College of
Physicians and Surgeons of Ontario shows that responsible journalism
exists in Canada. But not only did they not dig deep
enough, they omitted the government corruption end of it.

Patients win right to know

Tip: Share what you read.

Should doctors kill patients?

2009-03-12T08:37:00.000-07:00

Thank you for your letter

Re: Letters

Should doctors kill patients?

A. T. Kerigan

You have reached the most complete version of this article accessible.

We value your contribution. The cmaj.ca letters editor will review your submission, which if accepted, should be viewable within a few days.

If you have a problem with this process or other comments that do not pertain to the submission of electronic responses, then please use our feedback form.

Sincerely,
Editor cmaj.ca

Here is what your letter will look like online:

UpUrs!

Malcolm Everett

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The Doctors!

2009-03-11T17:27:00.000-07:00

Patients win right to know

The Toronto Star's courageous series on the College of Physicians and Surgeons of Ontario shows that responsible journalism exists in Canada.

TheStar.com/doctors

Tip: Share what you read.

www.SEXpatients.com

2009-03-11T17:01:00.000-07:00

www.SEXpatients.com

ENTER

sexpatients.com/

KDH C Difficile Communique

2009-03-10T23:41:00.000-07:00

Jordan E. H. MD (Medical Dolt)

705-567-9276
30 Second St E,
Kirkland Lake, ON,CA
P2N 1R1
RateMD's

kdhospitalscandal

Working Together To Meet YOUR Health Care Needs

Tip: Share what you read.

In the public eye!

2009-03-10T17:06:00.000-07:00

Jordan, Edward Henry Spiller, Mark Arthur

...physicians with a propensity for iatrogenic neglect and for

Alert on Ontario MD's

2009-03-10T17:02:00.000-07:00

Doctors Are The Leading Cause of Death in Canada!

2009-03-10T16:55:00.000-07:00

Ontario Doctors Exposed!!!

2009-03-10T16:30:00.000-07:00

Under Construction

Dr. A. B. Adegbite - Sudbury, ON Doctor Ratings | RateMDs.com

2009-03-10T16:06:00.000-07:00

Adegbite A B Dr Neurosurgeon
30 Durham St
Sudbury, ON
P3C 5E4
(705) 673-3130

ratemds.com

Andrew Adegbite, MD
Guillain-Barré Overlap Syndrome

Tip: Share what you read.

Mark Mager said:

2009-03-10T03:05:00.000-07:00

"Over the past few years, there have been many news reports of medical "professionals" taking part in the unauthorized removal, stealing and selling of body parts from unsuspecting patients and non-patients alike, dead or alive. The world-wide trade ( buying and selling ) and stealing of body parts is a multi-billion dollar "business". Everyone from nurses and ambulance drivers to doctors, surgeons and even morgue attendants and funeral home operators have been caught participating in these crimes. These illegal activities engaged in by medical "professionals" have taken place in many countries and on every continent. If any of you think this happens only in poor countries in Asia, Africa and South America, then you need to get your head out of the sand, give it a good shake and open your eyes. Since in the wealthy industrialized world we, in Canada, are near the bottom when it comes to "patient safety" ( medical errors, negligence, poor hospital hygiene etc.) why would we think that in other "problematic" areas of our healthcare ( like body parts stealing by medical "professionals" ) we would be so much safer? Sadly, the worlwide human body parts "business" is similar in many ways to the auto parts business. "Parts" are bought, sold, exchanged,stolen etc., and the "parts" dealers ( junkyard operators and some doctors ) have phone "hotlines" etc. to exchange "parts" information. Our "holier than thou" and "business as usual" medical institutions/hospitals etc. have lulled us into a false sense of security. The recent news reports about a doctor in Canada involved in stealing and/or buying and selling of hundreds of kidneys from other countries is just the tip of the developing iceberg. Just keep your eyes and ears open".

SOURCE: http://gussyupz.globalwhelming.com/?p=28

Brain Death Vs Brain Death Mimics

2009-03-09T18:02:00.000-07:00

Brain death is defined as the irreversible cessation of function of the entire brain with three specific criteria: 1) coma, 2) absent brainstem reflexes and 3) apnea. In addition to these clinical criteria, there are important prerequisites: 1) NO intoxication or poisoning, 2) NO core temperature greater than 32 degrees Celsius, 3) clinical or neuroimaging evidence of acute central nervous system catastrophe and 4) absence of confounding medical conditions such as severe electrolyte, acid-base, or endocrine disturbances.

Many physicians actively involved in the identification of brain death, if the truth be known, are unable to identify the requisite diagnostic components of brain death, and/or are unable to apply the criteria correctly. A second, or even third opinion should always be sought.

Canadian standards do not test function of the "entire brain"; does not assess subcortical function of structures such as the hypothalamus-pituitary axis. Many patients diagnosed as brain dead have intact neurohumoral control of hypothalamic-pituitary function, demonstrate normal hypothalamic mediated thermoregulatory control, and have intact autonomic function: they do not have hemodynamic collapse, they have physical findings such as bowel sounds, and are reported to have autonomic reflexes (tachycardia and hypertension) at the time of organ retrieval.

Given that current clinical testing does not assess subcortical brain function, ‘whole brain death’ cannot be conclusively identified at the bedside by using clinical criteria, and certainly not on the basis of a paltry CT. A CT scan is useful only in pretty severe cases, such as trauma, and also during the few days after an anoxic (lack of oxygen) brain injury. It’s useful in an emergency-room setting. But if the question is ischemic injury [brain damage caused by lack of blood/oxygen to part of the brain] you want an MRI and PET. For subsequent evaluation of brain injury, the CT is pretty useless unless there has been a massive stroke. In other words, being declared brain dead is a time consuming and detailed procedure, as it should be. It cannot be done in a meager few hours.

Brain death may be described as an esoteric creation of neurologists and neurosurgeons who are seeking to speed up the declaration of death for the purposes of an eye transplant, or a long awaited body part. The general practicing physician does not rely on those esoteric criteria in pronouncing a person dead." He uses the questionable "brain death criteria" based on a paltry CT as a pretext to operate and remove the eyes.

Many patients diagnosed as brain dead have hemodynamic instability. Many of these victims still have physical findings such as spontaneous breathing, including bowel sounds, and are reported to have autonomic (tachycardia and hypertension) in response to painful stimuli at the time of organ retrieval, suggesting a horrific death.

After the diagnosis of brain death, the focus of patient care shifts from interventions aimed at saving the patient's life to interventions aimed at maintaining viability of potentially transplantable organs

Brain death is what happens when ventilator support is discontinued. Condition called cerebral hypoxia or cerebral ischemia, is the direct result of oxygen deprivation to the brain cells. If proper balance is not restored or corrected, the heart and lungs may fail and the brain will literally begin to suffocate.

After several minutes of total oxygen deprivation, the brain may not be able to recover any meaningful function. The autonomic system controlling the heart and lungs fails next, leading to the ultimate cause of death, being oxygen deprivation of the brain cells, akin to "asphyxia".

It is never permissible to take any direct action designed to kill the patient. If a doctor deliberately let a patient die who was suffering from a curable treatable illness, the doctor would certainly be to blame for what he had done, just as he would be to blame if he had needlessly killed the patient. Charges against him would then be appropriate. If so, it would be no defense at all for him to insist that he didn't "do anything". He would have done something very serious indeed, for he let his patient die.

To allow or hasten a patient's demise to cover-up medical error or negligence is reprehensible. It is nothing short of murder since such an act or omission carries the intended consequence of the act or omission and therefore the mens rea or criminal intent. Negligence is where the accused ought to have foreseen the consequences of his actions, or inactions.

There are numerous reports of brain death declaration where the criteria were deliberately misrepresented, or obfuscated in an attempt to retrieve an organ for transplantation. The following is a perfect example.

The Arlene Berry Case

http://www.freewebs.com/medical_secrets/eFile.htm

In this case, the harried and hurried physicians did not rely on those esoteric criteria in pronouncing the patient "dead." They simply used the questionable "brain death criteria" (based on a paltry CT after withdrawal of ventillatory support) as a pretext to operate and remove their patient's eyes while she was still very much alive.

There have been many challenges to the several concepts of "brain death" and the means of their diagnosis worldwide (vide infra). Indeed, it seems that there is now an emerging consensus that "brain death" diagnosed by any of the protocols in current use worldwide is "not death."

As a safeguard in determining brain death a number of tests need to be carried out every 6 hours and recorded, the physicians performing this determination must not be part of a transplantation team. In some cases, 48 to 72 hours is required to evaluate brain death and a repeat examination with observation up to 24 hours is sometime needed. The length of time between serial examinations to declare brain death varies marginally from 6 to 72 hours.

Neurological and neurosurgical intensive care - Google Books Result

Brain Death Mimics

The effects of many sedative and anesthetic agents can closely mimic brain death. All the so-called major tranquilizers can mimic a death-like condition by causing reduced brain wave activity and lack of responsiveness. First, hypothermia may blunt brain stem responses when core temperatures are below 32°C and may mimic brain death below 27°C. Drug intoxication and neuromuscular blockade use are reversible causes of coma that can mimic brain death. Demyelinating polyradiculoneuropathy can also resemble brain death. Encephalopathy with peripheral neuropathy may falsely mimic brainstem death, as can viral encephalitis, brainstem encephalitis, rabies, including Locked-in syndrome

Drug intoxication may cause a deep physiologic depression that resembles brain death, and drug intoxication is the most common cause of coma of rapid onset which may mimic brain death. The examination can be marred by the effects of sedation.

Acute metabolic derangement and endocrine abnormalities can also mimic brain death. There may be other conditions that mimic brain death, or that provide examples of the mistaken diagnosis of brain death. Diabetes is the most common endocrine disorder. Severe hypermagnesemia can mimic brain death.

Some patients in this group have been proceeded to organ harvest with discontinuation of care with no conclusive evidence of permanent and irreversible loss of brain function, usually on the basis of a paltry CT, despite respiratory efforts (breathing spontaneously) and despite physical findings, ie 'bowel sounds' and automic reflexes with tachycardia and hypertension evident (which support normal subcortical functions), which is totally inconsistent with 'whole brain death'. The bald truth is that GBS patients with a spontaneous breathing pattern may require prolonged ventilation.

Fulminant cases of Guillain-Barre syndrome have been reported in which a rapid deterioration evolves to a clinical state "resembling brain death". GBS is one of the few neurological diseases whose clinical manifestations may mimic or appear to be identical to those in brainstem death, illustrating an extreme polyneuropathy.

As a safeguard in determining brain death a number of tests need to be carried out every 6 hours and recorded, the physicians performing this determination must not be part of a transplantation team.

Glasgow Coma Scale should be cautiously applied as a prognostic measure in patients with metabolic or toxic CNS insults. In some cases, 48 to 72 hours is required to evaluate brain death and a repeat examinations are required to increase the diagnostic yield with observation up to 24 hours is sometime needed. The length of time between serial examinations to declare brain death varies marginally from 6 to 72 hours.

A number of physiologic changes occur in GBS and are the very same physiologic changes seen in brain death: hemodynamic instability (response to respiratory challenges), endocrine abnormalities, hypothermia, coagulopathy, pulmonary dysfunction (with paralysis of breathing muscles), and electrolyte imbalances (related to Na negligence). Endocrine gland dysfunction in GBS is due to lack of tissue response.

While criteria for brain death vary, generally doctors evaluate brain stem reflexes such as response of the pupil to light, response to ice-water in the ear, and gag and swallowing reflexes. The patient may be taken off the ventilator to determine whether spontaneous breathing is present. In addition, the cause of the coma must be known, and factors that could mimic brain death, such as certain potentially anesthetizing or paralyzing drugs, must be eliminated.

Although many conditions can mimic brain death clinically upon examination, without excluding them you will KILL a person by homicide, or criminal negligence, despite the reversibility of brain damage.

Acute paralysis in Guillain-Barré syndrome (GBS) is related to a Na negligence.

2009-03-09T15:23:00.000-07:00

Acute paralysis in Guillain-Barré syndrome (GBS) is related to a Na negligence. It is associated with morbid and iatrogenic events involving IV salt and water, and channel blocking factor in the cerebrospinal fluid. Na, from natrium, is the chemical symbol/synonym for sodium.

Several neurologic disorders including Guillain-Barré syndrome (GBS) are associated with hyponatremia. Hyponatremia and its overly fast correction have major implications to the course of the underlying neurologic disease. Sudden onset of CNS dysfunction suggests acute onset of hyponatremia. Hyponatremia is reported to occur in up to one-third of patients with Guillain-Barré syndrome (GBS). Hyponatremia can be associated with low, normal, or high tonicity. An association of SIADH with GBS has been documented in a number reports.

The neurophysiological abnormalities seen in Guillain-Barré syndrome, chronic inflammatory demyelinating polyneuropathy, and multiple sclerosis, traditionally regarded as the result of demyelination, may also be explained by sodium channel dysfunction commonly regarded as electrolyte disorders. Disruption of the blood-brain barrier (BBB) following a rapid increase in serum sodium concentration is considered to play a critical role in the pathogenesis of osmotic demyelination.

Demyelination of the white matter is associated with breakdown of the blood brain barrier and the development of vasogenic edema. Vasogenic edema is the most common type of edema results from local disruption of the blood brain barrier. Once the barrier is breached, hydrostatic and osmotic forces work together to extravasate intravascular fluid. Once extravasated, fluid is retained outside the vasculature, mostly in the white matter of the brain, and within the bundles of myelinated axons of long tracts and commissural fibers.

Active demyelination can be detected with the use of gadolinium-enhanced MRI and usually indicates that there has been a breach in the blood -brain barrier. Alteration in the permeability of the blood-brain barrier may predispose.

Osmotic Demyelination Syndrome occurs with rapid correction of Hyponatremia. It is characterized by acute paralysis, dysarthria, dysphagia and other neurological symptoms.

Anti-GM1 antibody is one of the causative factors of conduction abnormality in GBS patients. Breakdown of the blood-nerve barrier, inflammation of the nerve roots and conduction block are leading symptoms. Antibodies are known to reversibly block the voltage-gated Na+-channels of nerve cells. The demyelination as a secondary event may actually be displaying a pronounced "blood-brain barrier breach".

A novel Na+ channel blocker may exhibit analgesic effects in some. The assumed mechanism of action to effect analgesia is the acute blocking of sodium channels. The role of analgesic and/or antiepileptic drugs with “sodium channel–blocking” properties are the very same drugs known to exacerbate autonomic dysfunction encountered in Guillain-Barré syndrome (GBS) and multiple sclerosis (MS). Autonomic dysfunction is a frequent and severe complication of Guillain-Barre syndrome, giving rise to the hypothesis that some GBS cases may therefore have a subclinical peripheral neuropathy of osmotic aetiology and that Guillain Barre Syndrome is also an iatrogenic disease.

Medical Stupidity

Epiphenom: The doctors who hasten death

The Arlene Berry Story

On Bad Doctors...

Questionable Doctors...

Truth & Justice Demanded

Doctors Forcing Patients to Sign Gag Orders: Undue influence

The Questionable Cases Of Dr. Charles Smith

The Questionable Cases Of Dr. Charles Smith

The Questionable Cases Of Dr. Charles Smith: A Review

From Cancer QUACK To Chief Coroner

Barry A. McLellan MD (Medical Dolt) LIAR

What Ever Happened to First Do No Harm?

A doctor is history's worst serial killer

How system helps shield bad doctors

Iatrogenic Stemetil Poisoning...!

Crimefest - Eh? Fake Doctors Notes?

Popping Super Growth Pills: The Case for Cognitive Enhancement?

Code 0: Patient Status Code 0

Don't Kill Me Doctor:

Wash Your Hands!

IATROGENIC

More Doctors Smoke Camels Than Any Other Cigarette

New Cigarette WARNING!

Cannabis Canada!

M.D. really stands for "Medical Dolt"!

Healthcare Ontario

DOCTORS' ETHICS?

The Iatrogenic Experience

< Truth / Justice / Patient Safety >It's a path

Silence Infers Enough!

Silence is the opposite of safety

* * * * * <Truth/Justice/ Patient Safety > * * * * *It's a path

Visit: www.patient-safety.com

On Bias

Only in Canada - Doctor ratings & reviews likely Censored!

mcrawford@searchstudio.net

Status Code 0

Rx for Injustice!

MedicalSecrets

Should doctors kill patients?

Conflict of Interest:

The Doctors!

www.SEXpatients.com

KDH C Difficile Communique

In the public eye!

Alert on Ontario MD's

Doctors Are The Leading Cause of Death in Canada!

Ontario Doctors Exposed!!!

Dr. A. B. Adegbite - Sudbury, ON Doctor Ratings | RateMDs.com

Mark Mager said:

Brain Death Vs Brain Death Mimics

Acute paralysis in Guillain-Barré syndrome (GBS) is related to a Na negligence.

< Truth / Justice / Patient Safety >
It's a path

* * * * * <Truth/Justice/ Patient Safety > * * * * *
It's a path